References
- Gaworzcwska E, Colman G. Changes in ihe pattern of infection caused by Streptococcus pyogenes. Epidemiol Infec. 1988; 100: 257–69.
- Shulman ST. Invasive and toxin related disease causcd by Group A streptococci. Ped Infect Dis J. 1991; 10: S28–31.
- Cone LA, Woodard DR, Schlievert PM, Tomory GS. Clinical and bactcriologic observations of a toxic shock-like syndrome due to Streptococcus pyogenes. N Engl J Med. 1987; 317: 146–9.
- Shaunak S, Wendon J, Monteil M, Gordon AM. Septic scarlet fever due to Streptococcus pyogenes cellulitis. QJ Med. 1988; 9: 921–5.
- Bisno AL. Group A streptococcal infections and acute rhcumatic fever. N Engl J Med. 1991; 325: 783–93.
- Efstratiou A. Outbreaks of human infection causcd by pyogenic streptococci of Lanccfield group C and G. J Med Microbiol. 1989; 29: 207–19.
- Skokbcrg K, Simonen H, Renkonen OV, Valtonen V. Beta-hacmolytic group A, B, C and G streptococcal septicaemia: a clinical study. Scand J Infect Dis. 1988; 20: 119–25.
- Stevens DL, Tanner MH, Winship J ct al. Severe group-A streptococcal infections associated with a toxic shock-like syndrome and scarlet fever toxin A.N Engl J Med. 1989, 321: 1–8.
- Deviere J. Bain H. Scrruys E. Thys JP. Septicaemia and meningitis caused by a group G strcptococus. J Infect. 1986; 13:285–7.
- Bone R. Gram positive organisms and sepsis. Arch Intern Med. 1994; 154:26–34.
- Tobias P, Mathison J, Ulcvitch R. A family of lipopolysaccharide binding proteins involved in the response to gram negative sepsis. J Biol Chem. 1988:263: 1379–81.
- Deighton C. Beta haemolytic streptococci and reactive arthritis in adults. Ann Rheum Dis. 1993; 52: 475–82.