References
- Lee DH, Bertran K, Kwon JH, et al. Evolution, global spread, and pathogenicity of highly pathogenic avian influenza H5Nx clade 2.3.4.4. J Vet Sci. 2017;18(S1):269–280.
- OFFLU. Avian influenza report March 2021 to September 2021. [cited 2022 Jan]. Available at https://www.offlu.org/wp-content/uploads/2021/10/OFFLU-Sept2021-AVIAN-Final.pdf.
- WHO. 2018 Protocols influenza virus detection. [cited 2022 Jan]. Available at https://www.who.int/influenza/gisrs_laboratory/Protocols_influenza_virus_detection_Nov_2018.pdf.
- Fereidouni SR, Harder TC, Gaidet N, et al. Saving resources: avian influenza surveillance using pooled swab samples and reduced reaction volumes in real-time RT-PCR. J Virol Methods. 2012;186(1-2):119–125.
- Zhou B, Donnelly ME, Scholes DT, et al. Single-reaction genomic amplification accelerates sequencing and vaccine production for classical and Swine origin human influenza a viruses. J Virol. 2009;83:10309–10313.
- Lee CY, An SH, Kim I, et al. Prerequisites for the acquisition of mammalian pathogenicity by influenza A virus with a prototypic avian PB2 gene. Sci Rep. 2017;7(1):10205.
- Lo FT, Zecchin B, Diallo AA, et al. Intercontinental spread of Eurasian highly pathogenic avian influenza A(H5N1) to Senegal. Emerg Infect Dis. 2022;28(1):234–237.
- Matrosovich M, Stech J, Klenk HD. Influenza receptors, polymerase and host range. Rev Sci Tech. 2009;28(1):203–217.
- Munier S, Larcher T, Cormier-Aline F, et al. A genetically engineered waterfowl influenza virus with a deletion in the stalk of the neuraminidase has increased virulence for chickens. J Virol. 2010;84:940–952.
- Stech O, Veits J, Abdelwhab ES, et al. The neuraminidase stalk deletion serves as major virulence determinant of H5N1 highly pathogenic avian influenza viruses in chicken. Sci Rep. 2015;5:13493.
- Herfst S, Schrauwen EJ, Linster M, et al. Airborne transmission of influenza A/H5N1 virus between ferrets. Science. 2012;336(6088):1534–1541.
- Xu C, Hu WB, Xu K, et al. Amino acids 473V and 598P of PB1 from an avian-origin influenza A virus contribute to polymerase activity, especially in mammalian cells. J Gen Virol. 2012;93(3):531–540.
- Youk SS, Leyson CM, Seibert BA, et al. Mutations in PB1, NP, HA, and NA contribute to increased virus fitness of H5N2 highly pathogenic avian influenza virus clade 2.3.4.4 in chickens. J Virol. 2020;95(5):e01675–20.
- Blaurock C, Blohm U, Luttermann C, et al. The C-terminus of non-structural protein 1 (NS1) in H5N8 clade 2.3.4.4 avian influenza virus affects virus fitness in human cells and virulence in mice. Emerg Microbes Infect. 2021;10(1):1760–1776.
- Fusaro A, Zecchin B, Vrancken B, et al. Disentangling the role of Africa in the global spread of H5 highly pathogenic avian influenza. Nat Commun. 2019;10(1):5310.