References
- Adamczyk S, Robin E, Simerabet M, Kipnis E, Tavernier B, Vallet B, Bordet R, Lebuffe G. 2010. Sevoflurane pre- and post-conditioning protect the brain via the mitochondrial K ATP channel. Br J Anaesth. 104(2):191–200. doi: https://doi.org/10.1093/bja/aep365
- Angus DC, Linde-Zwirble WT, Lidicker J, Clermont G, Carcillo J, Pinsky MR. 2001. Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care. Crit Care Med. 29(7):1303–1310. doi: https://doi.org/10.1097/00003246-200107000-00002
- Chen QX, Song SW, Chen QH, Zeng CL, Zheng X, Wang JL, Fang XM. 2014. Silencing airway epithelial cell-derived hepcidin exacerbates sepsis induced acute lung injury. Crit Care. 18(4):470. doi: https://doi.org/10.1186/s13054-014-0470-8
- Chuang CC, Shiesh SC, Chi CH, Tu YF, Hor LI, Shieh CC, Chen MF. 2006. Serum total antioxidant capacity reflects severity of illness in patients with severe sepsis. Crit Care. 10(1):R36. doi: https://doi.org/10.1186/cc4826
- Erbas O, Taskiran D. 2014. Sepsis-induced changes in behavioral stereotypy in rats; involvement of tumor necrosis factor-alpha, oxidative stress, and dopamine turnover. J Surg Res. 186(1):262–268. doi: https://doi.org/10.1016/j.jss.2013.08.001
- Fu Y, Hu X, Cao Y, Zhang Z, Zhang N. 2015. Saikosaponin a inhibits lipopolysaccharide-oxidative stress and inflammation in human umbilical vein endothelial cells via preventing TLR4 translocation into lipid rafts. Free Radic Biol Med. 89:777–785. doi: https://doi.org/10.1016/j.freeradbiomed.2015.10.407
- Galley HF. 2011. Oxidative stress and mitochondrial dysfunction in sepsis. Br J Anaesth. 107(1):57–64. doi: https://doi.org/10.1093/bja/aer093
- Guo RF, Ward PA. 2007. Role of oxidants in lung injury during sepsis. Antioxid Redox Signal. 9(11):1991–2002. doi: https://doi.org/10.1089/ars.2007.1785
- Gustot T. 2011. Multiple organ failure in sepsis: prognosis and role of systemic inflammatory response. Curr Opin Crit Care. 17(2):153–159. doi: https://doi.org/10.1097/MCC.0b013e328344b446
- Hawiger J, Veach RA, Zienkiewicz J. 2015. New paradigms in sepsis: from prevention to protection of failing microcirculation. J Thromb Haemost. 13(10):1743–1756. doi: https://doi.org/10.1111/jth.13061
- He H, Liu W, Zhou Y, Liu Y, Weng P, Li Y, Fu H. 2018. Sevoflurane post-conditioning attenuates traumatic brain injury-induced neuronal apoptosis by promoting autophagy via the PI3K/AKT signaling pathway. Drug Des Devel Ther. 12:629–638. doi: https://doi.org/10.2147/DDDT.S158313
- Hsing CH, Chou W, Wang JJ, Chen HW, Yeh CH. 2011. Propofol increases bone morphogenetic protein-7 and decreases oxidative stress in sepsis-induced acute kidney injury. Nephrol Dial Transplant. 26(4):1162–1172. doi: https://doi.org/10.1093/ndt/gfq572
- Keum YS. 2011. Regulation of the Keap1/Nrf2 system by chemopreventive sulforaphane: implications of posttranslational modifications. Ann N Y Acad Sci. 1229:184–189. doi: https://doi.org/10.1111/j.1749-6632.2011.06092.x
- Lavi S, Bainbridge D, D'Alfonso S, Diamantouros P, Syed J, Jablonsky G, Lavi R. 2014. Sevoflurane in acute myocardial infarction: a pilot randomized study. Am Heart J. 168(5):776–783. doi: https://doi.org/10.1016/j.ahj.2014.07.009
- Lee H, Park YH, Jeon YT, Hwang JW, Lim YJ, Kim E, Park SY, Park HP. 2015. Sevoflurane post-conditioning increases nuclear factor erythroid 2-related factor and haemoxygenase-1 expression via protein kinase C pathway in a rat model of transient global cerebral ischaemia. Br J Anaesth. 114(2):307–318. doi: https://doi.org/10.1093/bja/aeu268
- Lee I, Dodia C, Chatterjee S, Feinstein SI, Fisher AB. 2014. Protection against LPS-induced acute lung injury by a mechanism-based inhibitor of NADPH oxidase (type 2). Am J Physiol Lung Cell Mol Physiol. 306(7):L635–L644. doi: https://doi.org/10.1152/ajplung.00374.2013
- Li C, Ma D, Chen M, Zhang L, Zhang L, Zhang J, Qu X, Wang C. 2016. Ulinastatin attenuates LPS-induced human endothelial cells oxidative damage through suppressing JNK/c-Jun signaling pathway. Biochem Biophys Res Commun. 474(3):572–578. doi: https://doi.org/10.1016/j.bbrc.2016.04.104
- Lorente L, Martin MM, Almeida T, Abreu-Gonzalez P, Ferreres J, Sole-Violan J, Labarta L, Diaz C, Jimenez A. 2015. Association between serum total antioxidant capacity and mortality in severe septic patients. J Crit Care. 30(1):217 e7–12. doi: https://doi.org/10.1016/j.jcrc.2014.09.012
- Luo Y, Lu S, Dong X, Xu L, Sun G, Sun X. 2017. Dihydromyricetin protects human umbilical vein endothelial cells from injury through ERK and Akt mediated Nrf2/HO-1 signaling pathway. Apoptosis. 22(8):1013–1024. doi: https://doi.org/10.1007/s10495-017-1381-3
- Mantzarlis K, Tsolaki V, Zakynthinos E. 2017. Role of oxidative stress and mitochondrial dysfunction in sepsis and potential Therapies. Oxid Med Cell Longev. 2017:5985209. doi: https://doi.org/10.1155/2017/5985209
- Mayr FB, Yende S, Angus DC. 2014. Epidemiology of severe sepsis. Virulence. 5(1):4–11. doi: https://doi.org/10.4161/viru.27372
- Niture SK, Khatri R, Jaiswal AK. 2014. Regulation of Nrf2-an update. Free Radic Biol Med. 66:36–44. doi: https://doi.org/10.1016/j.freeradbiomed.2013.02.008
- Okamura T, Miura T, Iwamoto H, Shirakawa K, Kawamura S, Ikeda Y, Iwatate M, Matsuzaki M. 1999. Ischemic preconditioning attenuates apoptosis through protein kinase C in rat hearts. Am J Physiol. 277(5 Pt 2):H1997–H2001.
- Petersen Shay K, Moreau RF, Smith EJ, Hagen TM. 2008. Is alpha-lipoic acid a scavenger of reactive oxygen species in vivo? Evidence for its initiation of stress signaling pathways that promote endogenous antioxidant capacity. IUBMB Life. 60(6):362–367. doi: https://doi.org/10.1002/iub.40
- Qiu P, Dong Y, Li B, Kang XJ, Gu C, Zhu T, Luo YY, Pang MX, Du WF, Ge WH. 2017. Dihydromyricetin modulates p62 and autophagy crosstalk with the keap-1/Nrf2 pathway to alleviate ethanol-induced hepatic injury. Toxicol Lett. 274:31–41. doi: https://doi.org/10.1016/j.toxlet.2017.04.009
- Quoilin C, Mouithys-Mickalad A, Lecart S, Fontaine-Aupart MP, Hoebeke M. 2014. Evidence of oxidative stress and mitochondrial respiratory chain dysfunction in an in vitro model of sepsis-induced kidney injury. Biochim Biophys Acta. 1837(10):1790–1800. doi: https://doi.org/10.1016/j.bbabio.2014.07.005
- Shan Y, Yang F, Tang Z, Bi C, Sun S, Zhang Y, Liu H. 2018. Dexmedetomidine ameliorates the neurotoxicity of sevoflurane on the immature brain through the BMP/SMAD signaling pathway. Front Neurosci. 12:964. doi: https://doi.org/10.3389/fnins.2018.00964
- Singer M, Deutschman CS, Seymour CW, Shankar-Hari M, Annane D, Bauer M, Bellomo R, Bernard GR, Chiche JD, Coopersmith CM, et al. 2016. The third international consensus definitions for sepsis and septic shock (sepsis-3). JAMA. 315(8):801–810. doi: https://doi.org/10.1001/jama.2016.0287
- Stack C, Jainuddin S, Elipenahli C, Gerges M, Starkova N, Starkov AA, Jove M, Portero-Otin M, Launay N, Pujol A, et al. 2014. Methylene blue upregulates Nrf2/ARE genes and prevents tau-related neurotoxicity. Hum Mol Genet. 23(14):3716–3732. doi: https://doi.org/10.1093/hmg/ddu080
- Suzuki T, Yamamoto M. 2015. Molecular basis of the keap1-Nrf2 system. Free Radic Biol Med. 88(Pt B):93–100. doi: https://doi.org/10.1016/j.freeradbiomed.2015.06.006
- Tebay LE, Robertson H, Durant ST, Vitale SR, Penning TM, Dinkova-Kostova AT, Hayes JD. 2015. Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease. Free Radic Biol Med. 88(Pt B):108–146. doi: https://doi.org/10.1016/j.freeradbiomed.2015.06.021
- Victor VM, Espulgues JV, Hernandez-Mijares A, Rocha M. 2009. Oxidative stress and mitochondrial dysfunction in sepsis: a potential therapy with mitochondria-targeted antioxidants. Infect Disord Drug Targets. 9(4):376–389. doi: https://doi.org/10.2174/187152609788922519
- Watanabe K, Iwahara C, Nakayama H, Iwabuchi K, Matsukawa T, Yokoyama K, Yamaguchi K, Kamiyama Y, Inada E. 2013. Sevoflurane suppresses tumour necrosis factor-alpha-induced inflammatory responses in small airway epithelial cells after anoxia/reoxygenation. Br J Anaesth. 110(4):637–645. doi: https://doi.org/10.1093/bja/aes469
- Xu H, Zhao B, She Y, Song X. 2018. Dexmedetomidine ameliorates lidocaine-induced spinal neurotoxicity via inhibiting glutamate release and the PKC pathway. Neurotoxicology. 69:77–83. doi: https://doi.org/10.1016/j.neuro.2018.09.004
- Ye R, Yang Q, Kong X, Li N, Zhang Y, Han J, Xiong L, Liu X, Zhao G. 2012. Sevoflurane preconditioning improves mitochondrial function and long-term neurologic sequelae after transient cerebral ischemia: role of mitochondrial permeability transition. Crit Care Med. 40(9):2685–2693. doi: https://doi.org/10.1097/CCM.0b013e318258fb90
- Yeung JH, Ong GJ, Davies RP, Gao F, Perkins GD. 2012. Factors affecting team leadership skills and their relationship with quality of cardiopulmonary resuscitation. Crit Care Med. 40(9):2617–2621. doi: https://doi.org/10.1097/CCM.0b013e3182591fda
- Zenkov NK, Menshchikova EB, Tkachev VO. 2013. Keap1/Nrf2/ARE redox-sensitive signaling system as a pharmacological target. Biochemistry (Mosc). 78(1):19–36. doi: https://doi.org/10.1134/S0006297913010033