References
- Vígh B, Manzanoe, Silva MJ, Frank CL. The system of cerebrospinal fluid-contacting neurons. It supposed role in the non-synaptic signal transmission of the brain. Histol Histopathol. 2004;19:607–28.
- Wang MS, Zhang LC. Methodological comparison on the tracing CSF-CNs with HRP and CB-HRP. Acad Med. 1992;12:286–87.
- Zhang LC, Zeng YM, Ting J. The distributions and signaling directions of the cerebrospinal fluid contacting neurons in the parenchyma of a rat brain. Brain Res. 2003;989:1–8.
- Lu XF, Li YY, Zhang LC. Substance P in the cerebrospinal fluid-contacting nucleus contributes to morphine physical dependence in rats. Neurosci. Let. 2011;488:188–92.
- Du J, Yang XW, Zhang LC, Zeng YM. Expression of TRPM8 in the distal cerebrospinal fluid-contacting neurons in the brain mesencephalon of rats. Cerebrospinal Fluid Res. 2009;3:1–7.
- Xu C, Zhao ZJ, Wu TT. Distribution of TRPV1 in CSF contacting nucleus of rat brain parenchyma and its expression in neuropathic pain. J Neurol Neurophysiol. 2011;2:3.
- Lu XF, Geng XJ, Zhang LC. Substance P expression in the distal cerebrospinal fluid-contacting neurons and spinal trigeminal nucleus in formalin-induced the orofacial inflammatory pain in rats. Brain Res Bull. 2008;78:139–45.
- Waldmann R, Champigny G, Bassilana F. A proton-gated cation channel involved in acid-sensing. Nature. 1997;386:173–77.
- Krishtal O. The ASICs: signaling molecules? Modulators? Trends Neurosci. 2003;269:477–83.
- Deval E, Gasull X, Noel J, Salinas M, Baron A, Diochot S, . Acid sensing ion channels (ASICs): Pharmacology and implication in pain. Pharmacol Ther. 2010;128:549–58.
- Waldmann R, Lazdunski M. H+-gated cation channels: neuronal acid sensors in the NaC/DEG family of ion channels. Curr Opin Neurobiol. 1998;8:418–24.
- Sakai H, Lingueglia E, Champigny G. Cloning and functional expression of a novel degenerin-like Na+ channel gene in mammals. J Physiol. 1999;519:323–33.
- Yagi J, Wenk HN, Naves LA. Sustained currents through ASIC3 ion channels at the modest pH changes that occur during myocardial ischemia. Circ Res. 2006;99:501–9.
- Li WG, Xu TL. ASIC3 channels in multimodal sensory perception. ACS Chem Neurosci. 2011;2:26–37.
- Mamet J, Baron A, Lazdunski M. Proinflammatory mediators, stimulators of sensory neuron excitability via the expression of acid-sensing ion channels. J Neurosci. 2002;22:10662–70.
- Voilley N, Weille J, Mamet J. Nonsteroid anti-inflammatory drugs inhibit both the activity and the inflammation-induced expression of acid-sensing ion channels in nociceptors. J Neurosci. 2001;21:8026–33.
- Diochot S, Baron A, Rash LD, Deval E, Escoubas P, Scarzello SA. New sea anemone peptide, APETx2, inhibits ASIC3, a major acid-sensitive channel in sensory neurons. EMBO J. 2004;23:1516–25.
- Karczewski J, Spencer RH, Garsky VM. Reversal of acid-induced and inflammatory pain by the selective ASIC3 inhibitor, APETx2. Br J Pharmacol. 2010;161:950–60.
- Meng QY, Wang W, Chen XN. Distribution of acid-sensing ion channel 3 in the rat hypothalamus. Neuroscience. 2009;159:1126–34.
- Chen CH, Hsu YT, Chen CC. Acid-sensing ion channels in neurons of the rat suprachiasmatic nucleus. J Physiol. 2009;587:1727–37.
- Cao XL, Chen Q, Zhou H, Tang YH, Xu JG, Zheng Y. Expression of acid-sensing ion channels in neurons of trapezoid body and lateral paragigantocellular nuclei in rat brain, and effects of intermittent hypoxia on their expression. Sichuan Da Xue Xue Bao Yi Xue Ban. 2009;40:662–66.
- Wu WL, Lin YW, Min MY. Mice lacking Asic3 show reduced anxiety-like behavior on the elevated plus maze and reduced aggression. Genes Brain Behav. 2010;9:603–14.
- Wu WL, Cheng CF, Sun WH. Targeting ASIC3 for pain, anxiety, and insulin resistance. Pharmacol Ther. 2012;134:127–38.
- Lingueglia E. Acid-sensing ion channels in sensory perception. J Biol Chem. 2007;282:17325–29.
- Strittmatter M, Grauer M, Isenberg E. Cerebrospinal fluid neuropeptides and monoaminergic transmitters in patients with trigeminal neuralgia. Headache. 1997;37:211–16.
- Bianchi M, Martucci C, Ferrario P. Increased tumor necrosis factor-alpha and prostaglandin E2 concentrations in the cerebrospinal fluid of rats with inflammatory hyperalgesia: the effects of analgesic drugs. Anesth Analg. 2007;104:949–54.