References
- Messadi DV, Doung HS, Zhang Q et al. Activation of NFκB signal pathways in keloid fibroblasts. Arch. Dermatol. Res.296(3), 125–133 (2004).
- Duong HS, Zhang QZ, Le AD, Kelly AP, Kamdar R, Messadi DV. Elevated prolidase activity in keloids: correlation with type I collagen turnover. Br. J. Dermatol.154(5), 820–828 (2006).
- Igarashi A, Nashiro K, Kikuchi K et al. Connective tissue growth factor gene expression in tissue sections from localized scleroderma, keloid, and other fibrotic skin disorders. J. Invest. Dermatol.106(4), 729–733 (1996).
- Khoo YT, Ong CT, Mukhopadhyay A et al. Upregulation of secretory connective tissue growth factor (CTGF) in keratinocyte-fibroblast coculture contributes to keloid pathogenesis. J. Cell Physiol.208(2), 336–433 (2006).
- Xia W, Kong W, Wang Z et al. Increased CCN2 transcription in keloid fibroblasts requires cooperativity between AP-1 and SMAD binding sites. Ann. Surg.246(5), 886–895 (2007).
- Curran T, Franza BR Jr. Fos and Jun: the AP-1 connection. Cell55(3), 395–397 (1998).
- Kim CW, Suh SI, Sung SH, Lee IK, Lee KS. A transcriptional factor decoy against AP-1 suppresses TGF-β1-induced type I collagen gene expression in cultured keloid fibroblasts. J. Dermatol. Sci.37(1), 49–51 (2005).
- Fonseca C, Lindahl GE, Ponticos M et al. A polymorphism in the CTGF promoter region associated with systemic sclerosis. N. Engl. J. Med.357(12), 1210–1220 (2007).
- Leask A, Holmes A, Black CM, Abraham DJ. Connective tissue growth factor gene regulation.Requirements for its induction by transforming growth factor-β 2 in fibroblasts.J. Biol. Chem.278(15), 13008–13015 (2003).
- Xia W, Phan TT, Lim IJ, Longaker MT, Yang GP. Complex epithelial–mesenchymal interactions modulate transforming growth factor-β expression in keloid-derived cells. Wound Repair Regen.12(5), 546–556 (2004).
- Phan TT, Lim IJ, Aalami O et al. Smad3 signalling plays an important role in keloid pathogenesis via epithelial–mesenchymal interactions. J. Pathol.207(2), 232–242 (2001).
- Colwell AS, Yun R, Krummel TM, Longaker MT, Lorenz HP. Keratinocytes modulate fetal and postnatal fibroblast transforming growth factor-β and SMAD expression in co-culture. Plast. Reconstr. Surg.119(5), 1440–1445 (2007).
- Leask A, Sa S, Holmes A, Shiwen X, Black CM, Abraham DJ. The control of CCN2 (ctgf) gene expression in normal and scleroderma fibroblasts. Mol. Pathol.54(3), 180–183 (2001).