1,974
Views
13
CrossRef citations to date
0
Altmetric
Editorial

Myocardial fibrosis as a risk stratifier for sudden arrhythmic death

&
Pages 951-953 | Published online: 10 Jan 2014

References

  • Mehra R. Global public health problem of sudden cardiac death. J. Electrocardiol.40(Suppl. 6), S118–S122 (2007).
  • Garfinkel A, Chen PS, Walter DO et al. Quasiperiodicity and chaos in cardiac fibrillation. J. Clin. Invest.99(2), 305–314 (1997).
  • Weiss JN, Qu Z, Chen PS, Lin SF et al. The dynamics of cardiac fibrillation. Circulation112(8), 1232–1240 (2005).
  • Myerburg RJ, Mitrani R, Interian A Jr, Castellanos A. Interpretation of outcomes of antiarrhythmic clinical trials: design features and population impact. Circulation97(15), 1514–1521 (1998).
  • Rubart M, Zipes DP. Mechanisms of sudden cardiac death. J. Clin. Invest.115(9), 2305–2315 (2005).
  • Sovari AA. postinfarct cardiac remodeling and ventricular arrhythmias. In: Myocardial Ischemia; Causes, Symptoms, and Treatment. First Edition. Vukovik D, Kiyan V (Eds). Nova Science Publishers, NY, USA, 269–277 (2010).
  • Sovari AA, Morita N, Weiss JN, Karagueuzian HS. Serum transforming growth factor-β1 as a risk stratifier of sudden cardiac death. Med. Hypotheses71(2), 262–265 (2008).
  • Brooks A, Schinde V, Bateman AC, Gallagher PJ. Interstitial fibrosis in the dilated non-ischaemic myocardium. Heart89(10), 1255–1256 (2003).
  • Fielitz J, Hein S, Mitrovic V et al. Activation of the cardiac renin-angiotensin system and increased myocardial collagen expression in human aortic valve disease. J. Am. Coll. Cardiol.37(5), 1443–1449 (2001).
  • Wu TJ, Ong JJ, Hwang C et al. Characteristics of wave fronts during ventricular fibrillation in human hearts with dilated cardiomyopathy: role of increased fibrosis in the generation of reentry. J. Am. Coll. Cardiol.32(1), 187–196 (1998).
  • Morita N, Sovari AA, Xie Y et al. Increased susceptibility of aged hearts to ventricular fibrillation during oxidative stress. Am. J. Physiol. Heart Circ. Physiol.297(5), H1594–H1605 (2009).
  • Sovari AA. Oxidative stress promotes ventricular fibrillation at the early stages of hypertension: role of Ca2+/CaM kinase-II. Heart Rhythm7, 5–23 (2010).
  • Morita N, Lee JH, Bapat A et al. Glycolytic inhibition causes spontaneous ventricular fibrillation in aged hearts. Am. J. Physiol. Heart Circ. Physiol.301(1), H180–H191 (2011).
  • Miragoli M, Salvarani N, Rohr S. Myofibroblasts induce ectopic activity in cardiac tissue. Circ. Res.101(8), 755–758 (2007).
  • Ono N, Hayashi H, Kawase A et al. Spontaneous atrial fibrillation initiated by triggered activity near the pulmonary veins in aged rats subjected to glycolytic inhibition. Am. J. Physiol. Heart Circ. Physiol.292(1), H639–H648 (2007).
  • Schultz JJ, Witt SA, Glascock BJ et al. TGF-β1 mediates the hypertrophic cardiomyocyte growth induced by angiotensin II. J. Clin. Invest.109(6), 787–796 (2002).
  • Deten A, Holzl A, Leicht M, Barth W, Zimmer HG. Changes in extracellular matrix and in transforming growth factor beta isoforms after coronary artery ligation in rats. J. Mol. Cell. Cardiol.33(6), 1191–1207 (2001).
  • Li G, Borger MA, Williams WG et al. Regional overexpression of insulin-like growth factor-I and transforming growth factor-β1 in the myocardium of patients with hypertrophic obstructive cardiomyopathy. J. Thorac. Cardiovasc. Surg.123(1), 89–95 (2002).
  • Sanderson JE, Lai KB, Shum IO, Wei S, Chow LT. Transforming growth factor-β(1) expression in dilated cardiomyopathy. Heart86(6), 701–708 (2001).
  • Mitchell JA, Ventura HO, Mehra MR. Early recognition and treatment of hypertensive heart disease. Curr. Opin. Cardiol.20(4), 282–289 (2005).
  • Ban CR, Twigg SM. Fibrosis in diabetes complications: pathogenic mechanisms and circulating and urinary markers. Vasc. Health Risk Manage.4(3), 575–596 (2008).
  • Ban CR, Twigg SM. Fibrosis in diabetes complications: pathogenic mechanisms and circulating and urinary markers. Vasc. Health Risk Manage.4(3), 575–596 (2008).
  • Ito N, Ohishi M, Yamamoto K et al. Renin–angiotensin inhibition reverses advanced cardiac remodeling in aging spontaneously hypertensive rats. Am. J. Hypertens.20(7), 792–799 (2007).
  • Miric G, Dallemagne C, Endre Z, Margolin S, Taylor SM, Brown L. Reversal of cardiac and renal fibrosis by pirfenidone and spironolactone in streptozotocin-diabetic rats. Br. J. Pharmacol.133(5), 687–694 (2001).
  • Villarreal F, Zimmermann S, Makhsudova L et al. Modulation of cardiac remodeling by adenosine: in vitro and in vivo effects. Mol. Cell. Biochem.251(1–2), 17–26 (2003).
  • Maron MS. The current and emerging role of cardiovascular magnetic resonance imaging in hypertrophic cardiomyopathy. J. Cardiovasc. Transl. Res.2(4), 415–425 (2009).
  • Assomull RG, Prasad SK, Lyne J et al. Cardiovascular magnetic resonance, fibrosis, and prognosis in dilated cardiomyopathy. J. Am. Coll. Cardiol.48(10), 1977–1985 (2006).
  • Wu KC, Weiss RG, Thiemann DR et al. Late gadolinium enhancement by cardiovascular magnetic resonance heralds an adverse prognosis in nonischemic cardiomyopathy. J. Am. Coll. Cardiol.51(25), 2414–2421 (2008).
  • Iles L, Pfluger H, Lefkovits L et al. Myocardial fibrosis predicts appropriate device therapy in patients with implantable cardioverter-defibrillators for primary prevention of sudden cardiac death. J. Am. Coll. Cardiol.57(7), 821–828 (2011).

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.