References
- AlievGIs non-genetic Alzheimer’s disease a vascular disorder with neurodegenerative consequences?J Alzheimer’s Dis2002451316
- AlievGLiuJXuKApoE4 in mice induces age-dependent brain hypoperfusion, neuronal, glial and microvascular damage, and cognitive impairment, which can be prevented by feeding Acetyl-L-Carnitine and R-Lipoic Acid8th International Conference on Alzheimer’s and Parkinson’s Diseases AD/PD (Salzburg, Austria)2007Proceeding AD/PD ConferenceMonduzzi Editore, Bologna, Italyin press
- AlievGObrenovichMESeyidovaDExploring ischemia-induced vascular lesions and potential pharmacological intervention strategiesHistol Histopathol2005202617315578444
- AlievGSeyidovaDLambBTMitochondria and vascular lesions as a central target for the development of Alzheimer’s disease and Alzheimer disease-like pathology in transgenic miceNeurol Res2003a256657414503022
- AlievGSeyidovaDNealMLAtherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in aged transgenic miceAnn N Y Acad Sci2002a977456412480733
- AlievGShiJPerryGNeuronal mitochondrial abnormalities in yeast artificial chromosome (YAC) transgenic mice overexpressing amyloid precursor protein (APP)Society for Neuroscience Abstracts Book200030
- AlievGSmithMAObrenovichMERole of vascular hypoperfusion-induced oxidative stress and mitochondria failure in the pathogenesis of Alzheimer diseaseNeurotox Res2003b549150414715433
- AlievGSmithMASeyidovDThe role of oxidative stress in the pathophysiology of cerebrovascular lesions in Alzheimer’s diseaseBrain Pathol2002b12213511770899
- AlievGSmithMAVintersHMitochondria abnormalities mark vulnerable neurons in Alzheimer’s diseaseJ Neuropathol Exp Neurol199958511
- AliyevAChenSGSeyidovaDMitochondria DNA deletions in atherosclerotic hypoperfused brain microvessels as a primary target for the development of Alzheimer’s diseaseJ Neurol Sci2005229–23028592
- AliyevASeyidovaDRzayevNIs nitric oxide a key target in the pathogenesis of brain lesions during the development of Alzheimer’s disease?Neurol Res2004265475315265272
- AnnaháziAMracskóESüleZPre-treatment and post-treatment with alpha-tocopherol attenuates hippocampal neuronal damage in experimental cerebral hypoperfusionEur J Pharmacol20075712–3120817597609
- CashADAlievGSiedlakSLMicrotubule reduction in Alzheimer’s disease and aging is independent of tau filament formationAm J Pathol20031621623712707046
- ChoiSAKimEHLeeJYPreconditioning with chronic cerebral hypoperfusion reduces a focal cerebral ischemic injury and increases apurinic/apyrimidinic endonuc-lease/redox factor-1 and matrix metalloproteinase-2 expressionCurr Neurovasc Res20074899717504207
- De JongGIDe VosRASteurENCerebrovascular hypoperfusion: a risk factor for Alzheimer’s disease? Animal model and postmortem human studiesAnn N Y Acad Sci199782656749329681
- de la TorreJCHemodynamic consequences of deformed microvessels in the brain in Alzheimer’s diseaseAnn N Y Acad Sci199782675919329682
- de la TorreJCCritically attained threshold of cerebral hypoperfusion: the CATCH hypothesis of Alzheimer’s pathogenesisNeurobiol Aging2000213314210867218
- de la TorreJCAlzheimer’s disease: how does it start?J Alzheimer’s Dis2002a4497512
- de la TorreJCAlzheimer disease as a vascular disorder: nosological evidenceStroke2002b3311526211935076
- de la TorreJCHow do heart disease and stroke become risk factors for Alzheimer’s disease?Neurol Res200666374416945216
- de la TorreJCAlievGInhibition of vascular nitric oxide after rat chronic brain hypoperfusion: spatial memory and immunocytochemical changesJ Cereb Blood Flow Metab2005256637215703700
- FristonKJFrackowiakRSCerebral function in aging and Alzheimer’s disease: the role of PETElectroencephalogr Clin Neurophysiol Suppl199142355651915028
- HiraiKAlievGNunomuraAMitochondrial abnormalities in Alzheimer’s diseaseJ Neurosci20012130172311312286
- LambBABardelKAKulnaneLSAmyloid production and deposition in mutant amyloid precursor protein and presenilin-1 yeast artificial chromosome transgenic miceNat Neurosci19992695710412057
- KumarASchapiroMBHaxbyJVCerebral metabolic and cognitive studies in dementia with frontal lobe behavioral featuresJ Psychiatr Res199024971092213642
- KalbackWEshCCastañoEMAtherosclerosis, vascular amyloidosis and brain hypoperfusion in the pathogenesis of sporadic Alzheimer’s diseaseNeurol Res2004265253915265270
- LishmanovYShveraIUssovWThe effect of carotid endarterectomy on cerebral blood flow and cerebral blood volume studied by SPECTJ Neuroradiol199724155629297936
- MoreiraPISiedlakSLWangXAutophagocytosis of mitochondria is prominent in Alzheimer diseaseJ Neuropathol Exp Neurol2007665253217549012
- NunomuraAPerryGAlievGOxidative damage is the earliest event in Alzheimer diseaseJ Neuropathol Exp Neurol2001607596711487050
- RoherAEEshCKokjohnTACircle of willis atherosclerosis is a risk factor for sporadic Alzheimer’s diseaseArterioscler Thromb Vasc Biol20032320556214512367
- ShiJPerryGAlievGSerum amyloid P is not present in amyloid beta deposits of a transgenic animal modelNeuro Report199910322932
- ShiJPerryGBerridgeMSLabeling of cerebral amyloid beta deposits in vivo using intranasal basic fibroblast growth factor and serum amyloid P component in miceJ Nucl Med2002a4310445112163630
- ShiJSeyidovaDPerryGNeuronal mitochondria abnormalities in a transgenic mouse model overexpressing amyloid betaSociety for Neuroscience2002b29520
- SkoogIGustafsonDUpdate on hypertension and Alzheimer’s diseaseNeurol Res2006286051116945211
- TaguchiAMatsuyamaTMoriwakiHCirculating CD34-positive cells provide an index of cerebrovascular functionCirculation20041092972515184275