References
- Cohen JI , MeytsI. Editorial: EBV infection and human primary immune deficiencies. Front Immunol.11(130), (2020).
- Hoshino A , TanitaK, KandaKet al. High frequencies of asymptomatic Epstein–Barr virus viremia in affected and unaffected individuals with CTLA4 mutations. Clin. Immunol.195, 45–48 (2018).
- Kimura H , NishikawaK, HoshinoY, SofueA, NishiyamaY, MorishimaT. Monitoring of cell-free viral DNA in primary Epstein–Barr virus infection. Med. Microbiol. Immunol.188(4), 197–202 (2000).
- Yamamoto M , KimuraH, HironakaTet al. Detection and quantification of virus DNA in plasma of patients with Epstein–Barr virus-associated diseases. J. Clin. Microbiol.33(7), 1765–1768 (1995).
- Crawford DH . Biology and disease associations of Epstein–Barr virus. Philos. Trans. R Soc. Lond. B Biol. Sci.356(1408), 461–473 (2001).
- Chan KCA , WooJKS, KingAet al. Analysis of plasma Epstein–Barr virus DNA to screen for nasopharyngeal cancer. N. Engl. J. Med.377(6), 513–522 (2017).
- Gan YJ , SullivanJL, SixbeyJW. Detection of cell-free Epstein–Barr virus DNA in serum during acute infectious mononucleosis. J. Infect. Dis.170(2), 436–439 (1994).
- Kimura H , ItoY, SuzukiR, NishiyamaY. Measuring Epstein–Barr virus (EBV) load: the significance and application for each EBV-associated disease. Rev. Med. Virol.18(5), 305–319 (2008).
- Rochford R , MoormannAM. Burkitt's lymphoma. Curr. Top. Microbiol. Immunol.390(Pt 1), 267–285 (2015).
- Kimura H . EBV in T-/NK-cell tumorigenesis. Adv. Exp. Med. Biol.1045, 459–475 (2018).
- Cohen JI . Epstein–Barr virus infection. N. Engl. J. Med.343(7), 481–492 (2000).
- Fan H , GulleyML. Epstein–Barr viral load measurement as a marker of EBV-related disease. Mol. Diagn.6(4), 279–289 (2001).
- Hoshino Y , KimuraH, TanakaNet al. Prospective monitoring of the Epstein–Barr virus DNA by a real-time quantitative polymerase chain reaction after allogenic stem cell transplantation. Br. J. Haematol.115(1), 105–111 (2001).
- Holmes RD , Orban-EllerK, KarrerFR, RoweDT, NarkewiczMR, SokolRJ. Response of elevated Epstein–Barr virus DNA levels to therapeutic changes in pediatric liver transplant patients: 56-month follow up and outcome. Transplantation74(3), 367–372 (2002).
- Clave E , AgbalikaF, BajzikVet al. Epstein–Barr virus (EBV) reactivation in allogeneic stem-cell transplantation: relationship between viral load, EBV-specific T-cell reconstitution and rituximab therapy. Transplantation77(1), 76–84 (2004).
- Van Esser JW , NiestersHG, VanDer Holt Bet al. Prevention of Epstein–Barr virus-lymphoproliferative disease by molecular monitoring and preemptive rituximab in high-risk patients after allogeneic stem cell transplantation. Blood99(12), 4364–4369 (2002).
- Callan MF , StevenN, KrausaPet al. Large clonal expansions of CD8+ T cells in acute infectious mononucleosis. Nat. Med.2(8), 906–911 (1996).
- Kuwahara N , KodakaT, ZushiYet al. T-cell large granular lymphocytic (LGL) leukemia consists of CD4(+)/CD8(dim) and CD4(−)/CD8(+) LGL populations in association with immune thrombocytopenia, autoimmune neutropenia and monoclonal B-cell lymphocytosis. J. Clin. Exp. Hematop.59(4), 202–206 (2019).
- Balfour HH , HolmanCJ, HokansonKMet al. A prospective clinical study of Epstein–Barr virus and host interactions during acute infectious mononucleosis. J. Infect. Dis.192(9), 1505–1512 (2005).
- Langerak AW , VanDen Beemd R, Wolvers-TetteroILet al. Molecular and flow cytometric analysis of the Vbeta repertoire for clonality assessment in mature TCRalphabeta T-cell proliferations. Blood98(1), 165–173 (2001).
- Maini MK , GudgeonN, WedderburnLR, RickinsonAB, BeverleyPC. Clonal expansions in acute EBV infection are detectable in the CD8 and not the CD4 subset and persist with a variable CD45 phenotype. J. Immunol.165(10), 5729–5737 (2000).
- Hoshino Y , MorishimaT, KimuraH, NishikawaK, TsurumiT, KuzushimaK. Antigen-driven expansion and contraction of CD8+-activated T cells in primary EBV infection. J. Immunol.163(10), 5735–5740 (1999).
- Swets JA . ROC analysis applied to the evaluation of medical imaging techniques. Invest. Radiol.14(2), 109–121 (1979).
- Taylor GS , LongHM, BrooksJM, RickinsonAB, HislopAD. The immunology of Epstein–Barr virus-induced disease. Annu. Rev. Immunol.33, 787–821 (2015).
- Chijioke O , AzziT, NadalD, MünzC. Innate immune responses against Epstein–Barr virus infection. J. Leukoc. Biol.94(6), 1185–1190 (2013).
- Balfour HH Jr , OdumadeOA, SchmelingDOet al. Behavioral, virologic and immunologic factors associated with acquisition and severity of primary Epstein–Barr virus infection in university students. J. Infect. Dis.207(1), 80–88 (2013).
- Williams H , McaulayK, MacsweenKFet al. The immune response to primary EBV infection: a role for natural killer cells. Br. J. Haematol.129(2), 266–274 (2005).
- Lundell R , HartungL, HillS, PerkinsSL, BahlerDW. T-cell large granular lymphocyte leukemias have multiple phenotypic abnormalities involving pan-T-cell antigens and receptors for MHC molecules. Am. J. Clin. Pathol.124(6), 937–946 (2005).
- Morice WG , KurtinPJ, LeibsonPJ, TefferiA, HansonCA. Demonstration of aberrant T-cell and natural killer-cell antigen expression in all cases of granular lymphocytic leukemia. Br. J. Haematol.120(6), 1026–1036 (2003).
- Toga A , WadaT, SakakibaraYet al. Clinical significance of cloned expansion and CD5 down-regulation in Epstein–Barr Virus (EBV)-infected CD8+ T lymphocytes in EBV-associated hemophagocytic lymphohistiocytosis. J. Infect. Dis.201(12), 1923–1932 (2010).
- Sanikommu SR , ClementeMJ, ChomczynskiPet al. Clinical features and treatment outcomes in large granular lymphocytic leukemia (LGLL). Leuk. Lymphoma59(2), 416–422 (2018).