265
Views
6
CrossRef citations to date
0
Altmetric
INNER EAR

Therapeutic effect of adeno-associated virus (AAV)-mediated ADNF-9 expression on cochlea of kanamycin-deafened guinea pigs

, , , , &
Pages 1022-1029 | Received 01 Mar 2013, Accepted 20 Apr 2013, Published online: 15 Sep 2013

References

  • Rizzi MD, Hirose K. Aminoglycoside ototoxicity. Curr Opin Otolaryngol Head Neck Surg 2007;15:352–7.
  • Ruan F, Wang H, Gao W, Ji H, Xiao J, Wang F. Amelioration of nerve growth factor against noise-induced threshold shift: a transmission electron microscope observation. Zhonghua Er Bi Yan Hou Ke Za Zhi 2000;35:267–70.
  • Fritzsch B, Tessarollo L, Coppola E, Reichardt LF. Neurotrophins in the ear: their roles in sensory neuron survival and fiber guidance. Prog Brain Res 2004;146:265–78.
  • Dejda A, Sokolowska P, Nowak JZ. Neuroprotective potential of three neuropeptides PACAP, V IP and PH I. Pharmacol Rep 2005;57:307–20.
  • Ernfors P, Duan ML, ElShamy WM, Canlon B. Protection of auditory neurons from aminoglycoside toxicity by neurotrophin-3. Nat Med 1996;2:463–7.
  • Holtser-Cochav M, Divinski I, Gozes I. Tubulin is the target binding site for NAP-related peptides: ADNF-9, D-NAP, and D-SAL. J Mol Neurosci 2006;28:303–7.
  • Busciglio J, Pelsman A, Helguera P, Ashur-Fabian O, Pinhasov A, Brenneman DE, et al. NAP and ADNF-9 protect normal and Down's syndrome cortical neurons from oxidative damage and apoptosis. Curr Pharm Des 2007;13:1091–8.
  • Praetorius M, Hsu C, Baker K, Brough DE, Plinkert P, Staecker H. Adenovector-mediated hair cell regeneration is affected by promoter type. Acta Otolaryngol 2010;130:215–22.
  • Tang Y, Cummins J, Huard J, Wang B. AAV-directed muscular dystrophy gene therapy. Expert Opin Biol Ther 2010;10:395–408.
  • Zheng G, Zhu K, Wei J, Jin Z, Duan M. Adeno-associated viral vector-mediated expression of NT4-ADNF-9 fusion gene protects against aminoglycoside-induced auditory hair cell loss in vitro. Acta Otolaryngol 2010;131:136–41.
  • Kusunoki T, Cureoglu S, Schachern PA, Sampaio A, Fukushima H, Oktay MF, et al. Effects of aminoglycoside administration on cochlea elements in human temporal bones. Auris Nasus Larynx 2004;31:383–8.
  • Buszman E, Wrześniok D, Matusiński B. Ototoxic drugs. I. Aminoglycoside antibiotics. Wiad Lek 2003;56:254–9; (in Polish).
  • Sha SH, Schacht J. Antioxidants attenuate gentamicin-induced free radical formation in vitro and ototoxicity in vivo: D-methionine is a potential protectant. Hear Res 2000;142:34–40.
  • Kumral A, Yesilirmak DC, Sonmez U, Baskin H, Tugyan K, Yilmaz O, et al. Neuroprotective effect of the peptides ADNF-9 and NAP on hypoxic-ischemic brain injury in neonatal rats. Brain Res 2006;1115:169–78.
  • Glazner GW, Camandola S, Mattson MP. Nuclear factor-kB mediates the cell survival-promoting action of activity-dependent neurotrophic factor peptide-9. J Neurochem 2000;75:101–8.
  • Li Duan M, Bordet T, Mezzina M, Kahn A, Ulfendahl M. Adenoviral and adeno-associated viral vector mediated gene transfer in the guinea pig cochlea. Neuroreport 2002;13:1295–9.
  • Stone IM, Lurie DI, Kelley MW, Poulsen DJ. Adeno-associated virus-mediated gene transfer to hair cells and support cells of the murine cochlea. Mol Ther 2005;11:843–8.
  • Liu Y, Okada T, Sheykholeslami K, Shimazaki K, Nomoto T, Muramatsu S, et al. Specific and efficient transduction of cochlear inner hair cells with recombinant adeno2 associated virus type 3 vector. Mol Ther 2005;12:725–33.

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.