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Original Article

Aminoacetone Excretion in Porphyrias and in Chronic Lead Intoxication

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Pages 498-502 | Received 20 Jun 1964, Published online: 08 Jul 2009

References

  • De Matteis F., Rimington C. The biochemical disturbance in acute intermittent and experimental porphyria. Lancet 1962; I: 1332
  • Elliott W. H. Aminoacetone formation by Staphylococcus aureus. Biochem. J 1960; 74: 478
  • Gibson K. D., Laver W., Neuberger A. Initial stages in the biosynthesis of porphyrins. 2. The formation of δ -aminolaevulic acid from glycine and succinyl-coenzyme A by particles from chicken erythrocytes. Ibid 1958; 70: 71
  • Granick S., Urata G. Increase in activity of δ -aminolevulinic acid synthetase in liver mitochondria induced be feeding of 3,5-dicarbeth-oxy-1,4-dihydrocollidine. J. biol. Chem 1963; 238: 821
  • Haeger B. Urinary δ -aminolaevulic acid and porphobilinogen in different types of porphyria. Lancet 1958; II: 606
  • Haeger-Aronsen B. Studies on urinary excretion of d-aminolaevulic acid and other haempre-cursors in lead workers and lead-intoxicated rabbits. Scand. J. clin. Lab. Invest 1960; 12, Suppl. 47
  • Kikuchi G., Kumar A., Talmage Ph., Shemin D. The enzymatic synthesis of δ -aminolevulinic acid. J. biol. Chem 1958; 233: 1214
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  • Studnitz V.W., Unpubl
  • Tschudy D. P., Welland F. H., Collins A., Hunter G. Aminoacetone in acute intermittent porphyria. Lancet 1963; II: 660
  • Urata G., Granick S. Biosynthesis of α-aminoketones and the metabolism of aminoacetone. J. biol. Chem 1963; 238: 811
  • Waldenström J., Haeger-Aronsen B. Different patterns of human porphyria. Brit. med. J 1963; ii: 272
  • Welland F. H., Heliman E. S., Gaddis E. M., Collins A., Hunter G. W., Jr, Tschudy D. Factors affecting the excretion of porphyrin precursors by patients with acute intermittent porphyria. I. The effect of diet. Metabolism 1964; 13: 232

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