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Original Article

Helicobacter pylori Infection and Gastroduodenal Injury by Non-Steroidal Anti-Inflammatory Drugs

Pages 91-97 | Published online: 08 Jul 2009

References

  • McCarthy D M. Acid peptic disease in the elderly. Clinics Geriatric Med 1991; 7: 231–254
  • George L, Hyland L, Morgan A. Smoking does not contribute to duodenal ulcer relapse after helicobacter pylori eradication. Gastroenterology 1990; 98: A48
  • Martin D F, Montgomery E, Dobek A S, Patrissi G A, Peura D F. Campylobacter pylori, NSAIDS, and smoking: risk factors for peptic ulcer disease. Am J Gastroenterol 1989; 84: 1268–1272
  • McDonald W C. Correlation of mucosal histology and aspirin intake in chronic gastric ulcer. Gastroenterology 1973; 65: 381–389
  • McIntyre R LE, Irani M S, Piris J. Histological study of the effects of three anti-inflammatory preparations on the gastric mucosa. J Clin Pathol 1981; 34: 836–842
  • Peterson W L, Lee E L, Feldman M. Gastric campylobacter-like organisms in healthy humans: correlation with endoscopic appearance and mucosal histology. Gastroenterol 1986; 90: A1585
  • Gustavsson S, Malagelada J-R, Rosenblatt J E. Assessment of campylobacter-like organisms in the post-operative stomach, iatrogenic gastritis, and chronic gastroduodenal diseases: preliminary observations. Mayo Clin Proc 1987; 62: 265–268
  • Peterson W L, Lee E L, Feldman M. Relationship between campylobacter pylori and gastritis in healthy humans after administration of placebo or indo-methacin. Gastroenterology 1988; 95: 1185–1197
  • Laine L, Marin-Sorensen M, Weinstein W M. Is campylobacter pylori (CP) prevalence lower in gastric ulcers (GU's) because of non-steroid antiinflammatory drug use? A prospective evaluation. Gastroenterology 1988; 96: A282
  • Laine L, Marin-Sorensen M, Weinstein W M. Helicobacter pylori prevalence and mucosal injury in gastric ulcers: relationship to chronic nonsteroidal anti-inflammatory drug ingestion. Gastroenterology 1991; 100: A103
  • Doube A, Morris A. Nonsteroidal anti-inflammatory drug-induced dyspepsia—is campylobacter pyloridis implicated?. Br J Rheumatol 1988; 27: 110–112
  • Upadhyay R, Howatson A, McKinlay A, Danesh B JZ, Sturrock R D, Russell R I. Campylobacter pylori associated gastritis in patients with rheumatoid arthritis taking non-steroidal anti-inflammatory drugs. Br J Rheumatol 1988; 27: 113–116
  • Iglehart I W, Edlow D W, Mills L, Morrison S A, Hochberg M C. The presence of campylobacter pylori in non-steroidal anti-inflammatory drug associated gastritis. J Rheumatol 1989; 16: 599–603
  • Maxton D G, Srivastava E D, Whorwell P J, Jones D M. Do non-steroidal anti-inflammatory drugs or smoking predispose to helicobacter pylori infection?. Postgrad Med J 1990; 66: 717–719
  • Jones S TM, Clague R B, Eldridge J, Jones D M. Serological evidence of infection with helicobacter pylori may predict gastrointestinal intolerance to non-steroidal anti-inflammatory drug (NSAID) treatment in rheumatoid arthritis. Br J Rheumatol 1991; 30: 16–20
  • Caselli M, Pazzi R, LaCorte R, Aleotti A, Trevisani L, Stabellini G. Campylobacter-like organisms, non-steroidal anti-inflammatory drugs and gastric-lesions in patients with rheumatoid arthritis. Digestion 1989; 44: 101–104
  • Shallcross T M, Rathbone B J, Wyatt J I, Heatley R V. C. Pylori is the cause of ‘non-steroidal gastritis’. Klin Wochrnschr 1989; 67: 63, Suppl 18
  • Scherak O, Hirschl A M, Nemec H, Amann B, Kolarz G, Thumb N. NSAID associated gastritis and helicobacter pylori. J Rheumatol 1989; 16: 860–861
  • Scherak O, Hirschl A M, Nemec H, Amann B, Kolarz G, Thumb B. Helicobacter pylori and non-steroidal anti-rheumatics (GER). Dtsch Med Wochenschr 1990; 115: 1171–1175
  • Heresbach D, Raoul J L, Donnio P Y. Helicobacter pylori: a risk and severity factor in NSAID induced gastropathy. Gastroenterology 1991; 100: A82
  • Taha S D, Nakshabendi I, Boothman P. Chemical gastritis in patients receiving non-steroidal anti-inflammatory drugs—correlation with peptic ulceration [Abstract W61]. Gut 1991; 32
  • McCarthy D M. Non-steroidal anti-inflammatory drug-induced ulcers: management by traditional therapies. Gastroenterology 1989; 96: 662–674
  • Armstrong C P, Blower A L. Non-steroidal antiinflammatory drugs and life-threatening complications of peptic ulceration. Gut 1987; 28: 527–532
  • Whittle B JR, Esplugues J V. Ulcerogenic eicosanoids and related lipid mediators in gastric mucosal damage. Advances in drug therapy of gastrointestinal ulceration, A Garner, B JR Whittle. John Wiley & Sons, New York 1989; 165–188
  • Wallace J L. Lipid mediators of inflammation in gastric ulcer. Am J Physiol 1990; 258: G1–G11, (G.I. Liver Physiol 21)
  • Vaananen P M, Grisham M B, Wallace J L. Leukotriene D4 plays an important role in the pathogenesis of experimental NSAID gastropathy. Gastroenterology 1991; 100: A846
  • Parks D A. Oxygen radicals: mediators of gastrointestinal pathophysiology. Gut 1989; 30: 293–298
  • Granger D N, Hernandes L A, Grisham M B. Reactive oxygen metabolites: mediators of cell injury in the digestive system. Viewpoints Dig Dis (AGA) 1986; 18: 13–16
  • Wallace J L, Keenan C M, Granger D N. Gastric ulceration induced by non-steroidal anti-inflammatory drugs is a neutrophil dependent process. Am J Physiol 1990; 259: G462–G467, (GI/Liver Physiol 22)
  • Wallace J L, Arfors K-E, Webb-McKnight. A monoclonal antibody against the CD18 leukocyte adhesion molecule prevents indomethacin-induced gastric damage. Gastroenterol 1991; 100: 878–883
  • Vaananen P M, Meddings J B, Wallace J L. Role of active oxygen metabolites in experimental NSAID-gastropathy. Gastroenterology 1991; 100: A846
  • Slomiany B L, Bilski J, Sarosiek J. Campylobacter pyloridis degrades mucin and undermines gastric mucosal integrity. Biochem Biophys Res Comm 1987; 144: 307–314
  • Wallace J L. Gastric resistance to acid: is the ‘mucusbicarbonate barrier’ functionally redundant?. Am J Physiol 1989; 256: G31–G38, (GI/Liver Physiol 19)

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