Does the Stomach Adapt to Helicobacter pylori?

References

• Dixon M F, Sobala G M. Gastritis and duodenitis: the histo-pathological spectrum. Eur J Gastroenterol Hepatol 1992; 4: S 17–23, Suppl 2
   Web of Science ®Google Scholar
• Graham D Y. Pathogenic mechanisms leading to Helicobacter pylori-induced inflammation. Eur J Gastroenterol Hepatol 1992; 4: S9–16, Suppl 2
   Web of Science ®Google Scholar
• Evans D G, Evans D J, Jr, Graham D Y. Adherence and internalization of Helicobacter pylori by HEp-2 cells. Gastroenterology 1992; 102: 1557–67
   PubMed Web of Science ®Google Scholar
• Blaser M J. Hypotheses on the pathogenesis and natural history of Helicobacter pylori-'mduced inflammation. Gastroenterology 1992; 102: 720–7
   PubMed Web of Science ®Google Scholar
• Perez-Perez G I, Dworkin B M, Choclos J E, Blaser M J. Campylobacter pylori antibodies in humans. Ann Intern Med 1988; 109: 11–7
   PubMed Web of Science ®Google Scholar
• Mai U EH, Perez-Perez G I, Allen J B, Wahl S M., Blaser M J, Smith P D. Surface proteins from Helicobacter pylori exhibit chemotactic activity for human leukocytes and are present in gastric mucosa. J Exp Med 1992; 175: 517–25
   PubMed Web of Science ®Google Scholar
• Crabtree J E, Taylor J D, Wyatt J I, et al. Mucosal IgA recognition of Helicobacter pylori 120 kD protein, peptic ulceration and gastric pathology. Lancet 1991; 338: 332–4
   PubMed Web of Science ®Google Scholar
• Yoshimura H H, Evans D G, Graham D Y. H. pylori strains from duodenal ulcer patients differ at the genomic level from those from patient with simple gastritis. Entferm Digest 1990; 78: 22, Suppl 1
   Google Scholar
• Tytgat G NJ, Noach L A, Rauws E AJ. Helicobacter pylori. Eur J Gastroent Hepatol 1992; 4: 7–15, Suppl LS
   Web of Science ®Google Scholar
• Blaser N J. Pathogenesis of Helicobacter pylori-induced inflammation: a ‘slow’ bacterial infection?. Eur J Gastroenterol Hepatol 1992; 4: S17–19, Suppl 1
   Web of Science ®Google Scholar
• Tytgat G NJ, Noach L A, Rauws E AJ. Helicobacter pylori. Scand J Gastroenterol 1991; 26: 1–8, Suppl 187
   Web of Science ®Google Scholar
• Tytgat G NJ, Noach L A, Rauws E AJ. Helicobacter pylori infection and duodenal ulcer disease. Gastroenterol Clin N Am 1992, In press
   Web of Science ®Google Scholar
• Chittajulla R S, Ardill J ES, McColl K EL. The degree of hyp-ergastrinaemia induced by Helicobacter pylori is the same in duodenal ulcer patients and asymptomatic volunteers. Eur J Gastroenterol Hepatol 1992; 4: 49–59
   Web of Science ®Google Scholar
• Levi S, Haddad G, Ghosh P, Bearshall K, Playford R, Calam J. Campylobacter pylori and duodenal ulcers: the gastrin link. Lancet 1989; 1: 1167–8
   PubMed Web of Science ®Google Scholar
• Smith J TL, Pounder R E, Nwokolo C U, et al. Inappropriate hypergastrinaemia in asymptomatic healthy subjects infected with Helicobacter pylori. Gut 1990; 31: 522–5
   PubMed Web of Science ®Google Scholar
• Fiddian-Green R G, Bank S, Mark I M, Louw J H. Maximum acid output and position of peptic ulcers. Lancet 1976; 2: 1370–3
   PubMed Web of Science ®Google Scholar
• Teichmann R K, Pratschke E, Grab J, Hammer C, Brendel W. Gastrin release by interleukin-2 and T-interferon in vitro. Can J Physol Pharmacol 1986; 64, Suppl:62
   Google Scholar
• Murthy U K, Linscheer R, Cho C. The hypergastrinemia in Helicobacter pylori (Hp)-gastritis is due to a decrease in antral D cell density and D:E cell ratio. Gastroenterology 1992; 102: A130
   Google Scholar
• Haruma K, Sumii K, Okamoto S, et al. Helicobacter pylori infection causes low antral somatostatin content: pathogenesis of inappropriate hypergastrinemia. Gastroenterology 1992; 102: A80
   Google Scholar
• Levi S, Bearshall K, Swift I, et al. Antral Helicobacter pylori hypergastrinaemia and duodenal ulcers: effect of eradicating the organisms. Br Med J 1989; 299: 1504–5
   PubMed Web of Science ®Google Scholar
• Oderda G, Holton J, Altare F, Vaira D, Ainley C, Ansaldi N. Amoxycillin plus tinidazole for Campylobacter pylori gastritis in children: assessment by serum IgG antibody, pepsinogen 1, and gastrin levels. Lancet 1989; 1: 690–2
   PubMed Web of Science ®Google Scholar
• Graham D Y, Opekun A, Ginger M L, Evans D J, Klein P D, Evans D G. Ablation of exaggerated meal-stimulated gastrin release in duodenal ulcer patients after clearance of Helicobacter (Campylobacter) pylori infection. Am J Gastroenterol 1990; 85: 394–8
   PubMed Web of Science ®Google Scholar
• McColl K L, Fullarton G M, El Nujumi A N, MacDonald M L, Dahil S, Hilditch T E. Serum gastrin and gastric acid status one and seven months after eradication of Helicobacter pylori in duodenal ulcer patients. Gut 1990; 31: A601
   Web of Science ®Google Scholar
• Prewett E J, Hudson M, Nwokolo C U, Sawyerr A M, Pounder R E. Return of nocturnal intragastric acidity after a dosing with either ranitidine or omeprazole. Gastroenterology 1991; 100: 873–7
   PubMed Web of Science ®Google Scholar
• Peterson W, Lee E, Skoglund M. The role of Campylobacter pyloridis in epidemic gastritis with hypochlorhydria. Gastroenterology 1987; 92: A1575
   Google Scholar
• Graham D Y, Smith J L, Alpert L C, Yoshimura H H. Iatrogenic Campylobacter pylori infection is a cause of epidemic ach-lorhydria. Am J Gastroenterol 1988; 83: 974–80
   PubMed Web of Science ®Google Scholar
• Steer H. Mucosa-related bacteria in the stomach. Lancet 1984; 2: 528
   PubMed Web of Science ®Google Scholar
• Morris A, Nicholson G. Ingestion of Campylobacter pyloridis causes gastritis and raised fasting gastric pH. Am J Gastroenterol 1987; 82: 192–9
   PubMed Web of Science ®Google Scholar
• Barthel J S, Westblom T U, Havey A D, Gonzales F, Everett E D. Gastritis and Campylobacter pylori in healthy, asymptomatic volunteers. Arch Intern Med 1988; 148: 1149–51
   PubMedGoogle Scholar
• Cave D R, Vargas M. Effect of a Campylobacter pylori protein on acid secretion by parietal cells. Lancet 1989; 2: 187–9
   PubMed Web of Science ®Google Scholar
• McColl K EI, Fullaston G M, Chittajulla R S, et al. Plasma gastrin, daytime intra gastric pH and nocturnal acid output before and at 1 and 7 months following eradication of Helicobacter pylori in the duodenal ulcer subjects. Scand J Gastroenterol 1991; 26: 339–46
   PubMed Web of Science ®Google Scholar
• Rathbone B. Serological response to Helicobacter pylori. Eur J Gastroenterol Hepatol 1992; 4: S21–3, Suppl 1
   Web of Science ®Google Scholar
• Bontkes J H, Veenendaal R A, Peiia A S, et al. IgG subclass response to Helicobacter pylori in patients with chronic active gastritis and duodenal ulcer. Scand J Gastroenterol 1992; 27: 129–33
   PubMed Web of Science ®Google Scholar
• Kekki M, Sipponen P, Siurala M. Progression of antral and body gastritis in patients with active and healed duodenal ulcer and duodenitis. Scand J Gastroenterol 1984; 19: 382–8
   PubMed Web of Science ®Google Scholar
• Dixon M F. Helicobacter pylori and peptic ulceration: histological aspects. J Gastroenterol Hepatol 1991; 6: 125–30
   PubMed Web of Science ®Google Scholar
• Wyatt J I, Rathbone B J, Dixon M F, Heatly R V. Campylobacter pyloridis and acid induced gastric metaplasia in the pathogenesis of duodenitis. J Clin Pathol 1987; 40: 841–8
   PubMed Web of Science ®Google Scholar
• Maaroos H I, Salupere V, Uibo R, Kekki M, Sipponen P. Seven year follow-up study of chronic gastritis in gastric ulcer patients. Scand J Gastroenterol 1985; 20: 198–204
   PubMed Web of Science ®Google Scholar
• Kimura K. Chronological transition of the fundic-pyloric border determined by stepwise biopsy of the lesser and greater curvatures of the stomach. Gastroenterology 1972; 63: 584–92
   PubMed Web of Science ®Google Scholar
• Correa P. Chronic gastritis: A clinico-pathological classification. Am J Gastroenterol 1988; 83: 504–9
   PubMed Web of Science ®Google Scholar
• Sipponen P, Seppala K, Aarynen M, Helske T, Kettunen P. Chronic gastritis and gastroduodenal ulcer: a case control study on risk of coexisting duodenal or gastric ulcer in patients with gastritis. Gut 1989; 30: 922–9
   PubMed Web of Science ®Google Scholar
• Craanen M E, Dekker W, Blok P, Ferweda J, Tytgat G NJ. Intestinal metaplasia and Helicobacter pylori: an endoscopic bioptic study of the gastric antrum. Gut 1992; 33: 16–20
   PubMed Web of Science ®Google Scholar
• Dixon M F. Progress in the pathology of gastritis and duodenitis. Gastrointestinal pathology-current topics in pathology, G T Williams. Springer Verlag, Heidelberg 1990; Vol. 81: 1–40
   Google Scholar
• Scott N, Lansdown M, Diament R, et al. Helicobacter gastritis and intestinal metaplasia in a gastric cancer family [letter]. Lancet 1990; 335: 728
   PubMed Web of Science ®Google Scholar
• Engstrand L, Scheynius A, Pahlson C, Grimelius L, Schwan A, Gustavsson S. Association of Campylobacter pylori with induced expression of class II transplantation antigens on gastric epithelial cells. Infect Immun 1989; 57: 827–32
   PubMed Web of Science ®Google Scholar
• Rathbone B J, Wyatt J I, Trejdosiewicz L K, et al. Mucosal T cell subsets in normal gastric antrum and C. pylori-associated chronic gastritis. Gut 1988; 29: A1438
   Web of Science ®Google Scholar
• Wyatt J I, Rathbone B J. Immune response of the gastric mucosa to Campylobacter pylori. Scand J Gastroenterol 1988; 23: 44–9, Suppl 142
   Web of Science ®Google Scholar
• Engstrand L, Pahlson C, Scheynius A. An increased number of gamma/delta T cells and gastric epithelial cell expression of the gro EL stress-protein homologue in Helicobacter pylori associated gastritis of the antrum. Am J Gastroenterol 1991; 88: 976–80
   Google Scholar
• Evans D J, Jr, Evans D G, Engstrand L, Graham D Y. The 62-K urease-associated protein of Helicobacter pylori is a heat-shock protein. Ital J Gastroenterol 1991; 23: 30, Suppl 2
   PubMedGoogle Scholar
• Dunn B E, Roop H, Sung C, Perez-Perez G, Blaser M. Identification of a heatshock protein homolog in Helicobacter pylori [abstract B145]. Proceedings of the 91st Annual Meeting of the Am Soc Microbiol. Washington, DC 1991
   Google Scholar
• Papadimitrou C S, Ioachim-Velogianni E E, Tsianos E B, Montsopoulos H M. Epithelial HLD-DR expression and lymphocyte subsets in gastric mucosa in type B chronic gastritis. Virchows Arch [Pathol Anat] 1988; 413: 197–204
   Google Scholar
• Hatz R A, Brooks W P, Bayerdorfer E, Enders E, Kramling H J. The reactivity of peripheral blood lymphocytes to Helicobacter pylori antigens. Rev Esp Enf Dig 1990; 78: S76–7
   PubMedGoogle Scholar
• Karttunen R, Andersson G, Poikonenk K, et al. Helicobacter pylori induces lymphocyte activation in peripheral blood cultures. Clin Exp Immunol 1990; 82: 485–8
   PubMed Web of Science ®Google Scholar