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Original Article

Nitric Oxide Contributes to Multiorgan Oxidative Stress in Acute Experimental Pancreatitis

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Pages 943-948 | Received 18 Oct 1993, Accepted 31 Jan 1994, Published online: 08 Jul 2009

References

  • Sanfey H. Oxygen free radicals in experimental pancreatitis. The pathogenesis of pancreatitis, J M. Braganza. Manchester University Press, Manchester 1991; 53–65
  • Schoenberg M H., Büchler M, Beger H G. The role of oxygen radicals in experimental acute pancreatitis. Free Radic Biol Med 1992; 12: 515–22
  • Dabrowski A, Gabryelewicz A, Wereszczynska-Siemiatkowska U, Chyczewski L. Oxygen-derived free radicals in cerulein-induced acute pancreatitis. Scand J Gastroenterol 1988; 23: 1245–9
  • Dabrowski A, Chwiecko M. Oxygen radicals mediate depletion of pancreatic sulfhydryl compounds in rats with cerulein-induced acute pancreatitis. Digestion 1990; 47: 15–9
  • Dabrowski A, Gabryelewicz A. Oxidative stress–an early phenomenon characteristic of acute experimental pancreatitis. Int J Pancreatol 1992; 12: 193–9
  • Moncada S. Nitric oxide gas: mediator, modulator, and pathophysiologic entity. J Lab Clin Med 1992; 120: 187–91
  • Nathan C. Nitric oxide as a secretory product of mammalian cells. FASEB J 1992; 6: 3051–64
  • Stark M E., Szurszewski J H. Role of nitric oxide in gastrointestinal and hepatic function and disease. Gastroenterology 1992; 103: 1928–49
  • Jorens P G., Vermeire P A., Herman A G. L-arginine-dependent nitric oxide synthase: a new metabolic pathway in the lung and airways. Eur Respir i 1993; 6: 258–66
  • Lipton S A., Choi Y-B, Pan Z-H, et al. A redox-based mechanism for the neuroprotective and neurodestructive effects of nitric oxide and related nitroso-compounds. Nature 1993; 364: 626–32
  • Aho H J., Koskensalo S M-L, Nevalainen T J. Experimental pancreatitis in the rat. Sodium taurocholate-induced acute haem-orrhagic pancreatitis. Scand J Gastroenterol 1980; 15: 411–6
  • Salaris S C., Babbs C F. A rapid, widely applicable screen for drugs that suppress free radical formation in ischaemia/ reperfusion. J Pharmacol Methods 1988; 20: 335–45
  • Ellman G L. Tissue sulfhydryl groups. Arch Biochem Biophys 1959; 82: 70–7
  • Lowry O H., Rosenbrough N J., Farr A L., et al. Protein measurement with the Folin phenol reagent. J Biol Chem 1951; 193: 265–75
  • Caraway W T. A stable starch substrate for the determination of amylase in serum and other body fluids. Am J Clin Pathol 1959; 33: 97–108
  • Willemer S, Feddersen C O., Karges W, et al. Lung injury in acute experimental pancreatitis in rats. Int J Pancreatol 1991; 8: 305–21
  • Archer S L., Cowan N J. Acetylcholine causes endothelium dependent vasodilatation but does not stimulate nitric oxide production by rat pulmonary arteries or elevate endothelial cytosoli calcium concentrations. Circ Res 1991; 68: 1569–81
  • Shimizu I, Wada S, Okahisa T, et al. Radioimmunoreactive plasma bradykinin levels and histological changes during the course of cerulein-induced pancreatitis in rats. Pancreas 1993; 8: 220–5
  • Rinderknecht H. Acute necrotizing pancreatitis and its complications: an excessive reaction of natural defence mechanisms?. The pathogenesis of pancreatitis, J M. Braganza. Manchester University Press, Manchester 1991; 86–100
  • Tani S, Itoh H, Okabayashi Y, et al. New model of acute necrotizing pancreatitis induced by excessive doses of arginine in rats. Dig Dis Sci 1990; 35: 367–74

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