References
- Quargliarello V, Scheld W M. Bacterial meningitis: pathogenesis, pathophysiology, and progress. N Engl J Med 1992; 327: 864–872
- Tuomanen E, Tomasz A, Hengstler B, Zak O. The relative role of bacterial cell wall and capsule in the induction of inflammation in pneumococcal meningitis. J Infect Dis 1985; 151: 535–540
- Tuomanen E, Liu H, Hengstler B, Zak O, Tomasz A. The induction of meningeal inflammation by components of the pneumococcal cell wall. J Infect Dis 1985; 151: 859–868
- Leist T P, Frei K, Kam-Hansen S, Zinkernagel R M, Fontana A. Tumor necrosis factor α in cerebrospinal fluid during bacterial, but not viral, meningitis: evaluation in murine model infections and in patients. J Exp Med 1988; 167: 1743–1748
- Waage A, Halstensen A, Shalaby R, Brandtzaeg P, Kierulf P, Espevik T. Local production of tumor necrosis factor α, interleukin 1, and interleukin 6 in meningococcal meningitis: relation to the inflammatory response. J Exp Med 1989; 170: 1859–1867
- Lebel M H, Freij B J, Syrogiannopoulos G A, Chrane D F, Hoyt M J, Stewart S M, Kennard B D, Olsen K D, McCracken G H, Jr. Dexamethasone therapy for bacterial meningitis. N Engl J Med 1988; 319: 964–971
- The Guillain–Barré Syndrome Study Group. Plasmaphoresis and acute Guillain–Barré syndrome. Neurology 1985; 35: 1096–1104
- Hughes R AC, Newsom-Davis J M, Perkin G D, Pierce J M. Controlled trial of prednisolone in acute polyncuropathy. Lancet 1978; ii: 750–753
- Rother S, Knoblauch K D, Kirschfink M. Filtration of liquor cerebrospinals (CSF-filtration): technical concept and filter performance under in vitro conditions. Neuropsychology 1995; 9: 82–85
- Hülser P J, Wiethölter H, Wollinsky K H. Liquorpheresis eliminates blocking factors from cerebrospinal fluid in polyradiculoneuritis (Guillain–Barré syndrome). Eur Arch Psychiatry Clin Neurosci 1991; 241: 69–72
- Wollinsky K H, Weindler M, Hülser P J, Matzek N, Mehrkens H H, Kornhuber H H. Liquorpheresis (CSF-filtration): an effective treatment in acute and chronic severe autoimmune polyradiculoneuritis (Guillain–Barré syndrome). Eur Arch Psychiatry Clin Neurosci 1991; 241: 73–76
- Brinkmeier H, Wollinsky K H, Hülser P J, Seewald M J, Mehrkens H H, Kornhuber H H, Rüdel R. The acute paralysis in Guillain–Barré syndrome is related to a Na+ channel blocking factor in the cerebrospinal fluid. Pflügers Arch 1992; 421: 552–557
- Pfausler B, Auckenthaler A, Grubwieser G, Vollert-Rogenhofer H, Schmutzhard E. Miller–Fischer-Syndrom – erfolgreiche Therapie mit Liquorfiltration – ein Fallbericht. Neuropsychology 1994; 8: 41
- Wollinsky K H, Hülser P J, Mauch E, Mehrkens H H, Kornhuber H H. Liquorpherese bei 10 Patienten mit Multipler Sklerose. Verh Dtsch Gesellsch Neurol 1992; 7: 444–445
- Pfausler B, Bösch S, Grubwieser G, Vollert H, Greil R, Hagn C, Schmutzhard E. Multimodal therapy in life-threatening cerebral lupus erythematosus: the benefit of cerebrospinal fluid pheresis. Int Arch Allergy Immunol 1995; 107: 592–594
- Pfausler B, Grubwieser G, Bösch S, Vollert H, Herold M, Schmutzhard E. Cerebrospinal fluid-filtration reduces TNF in bacterial meningitis-CSF. Eur J Neurol, (in press)
- Schmutzhard E, Grubwieser G, Pfausler B. Liquorfiltration: eine adjuvante therapeutische Strategie bei der bakteriellen Meningitis. Neuropsychology 1995; 9: 104–105
- Starmark J E, Stälhammar D, Holmgren E. The reaction level scale (RLS 85). Manual and guidelines. Acta Neurochir 1988; 91: 12–20
- Wollinsky K H, Hülser P J, Brinkmeier H, Mehrkens H H, Kornhuber H H, Rüdel R. Klinische Erfahrungen mit der CSF-Filtration bei Guillain–Barré-Syndrom, chronisch inflammatorischer demyelinisierender Polyneuropathie und multipler Sklerose. Neuropsychology 1995; 9: 95–99
- van Furth A M, Seijmonsbergen E M, Langermans J AM, Groeneveld P HP, de Bel C E, van Furth R. High levels of interleukin 10 and tumor necrosis factor a in cerebrospinal fluid during the onset of bacterial meningitis. Clin Infect Dis 1995; 21: 220–222
- Rydberg J, Miörner H, Chandramuki A, Lantz M. Assessment of a possible imbalance between tumor necrosis factor (TNF) and soluble TNF receptor forms in tuberculosis infection of the central nervous system. J Infect Dis 1995; 172: 301–304