REFERENCES
- Mehta SR, Afenyi-Annan A, Byrns PJ, Lottenberg R. Opportunities to improve outcomes in sickle cell disease. Am Fam Physician. 2006;74(2):303–310.
- Jeremiah ZA. Abnormal haemoglobin variants, ABO and Rh blood groups among student of African descent in Port Harcourt, Nigeria. Afr Health Sci. 2006;6(3):177–181.
- El-Hazmi MA, Warsy AS. Appraisal of sickle-cell and thalassaemia genes in Saudi Arabia. East Mediterr Health J. 1999;5(6):1147–1153.
- Okpala I. The intriguing contribution of white blood cells to sickle cell disease–a red cell disorder. Blood Rev. 2004;18(1):65–73.
- Lard LR, Mul FP, de Hass M, Roos D, Duits AJ. Neutrophil activation in sickle cell disease. J Leuk Biol. 1999;66(3):411–415.
- Janeway C, Travers P, Walport M, Shlomchik M. Immunobiology: The Immune System in Health and Disease, 6th ed. New York: Garland Publishing Inc., 2005.
- Belcher JD, Marker PH, Weber JP, Hebbel RP, Vercellotti GM. Activated monocytes in sickle cell disease: potential role in the activation of vascular endothelium and vaso-occlusion. Blood. 2000;96(7):2451–2459.
- Bunn HF. Pathogenesis and treatment of sickle cell disease. N Engl J Med. 1997;337(11):762–769.
- Platt OS, Brambilla DJ, Rosse WF. Mortality in sickle cell disease: life expectancy and risk factors for early death. N Engl J Med. 1994;330(23):1639–1644.
- Sawyer DW, Donowitz GR, Mandell GL. Polymorph nuclear neutrophils: an effective antimicrobial force. Rev Infect Dis. 1989;11(Suppl. 7):S1532–S1544.
- Philip JR. Basic Principle in Clinical Flow Cytometry. Chicago: ASCP Press, 1994.
- Rothe G, Oser A, Valet G. Dihydrorhodamin 123: a new flow cytometric indicator for respiratory burst activity in neutrophil granulocytes. Naturwissenschaften. 1988;75(7):354–355.
- Sanz ML, Maselli JP, Gamboa PM, Oehling A, Diéguez I, de Weck AL. Flow cytometric basophil activation test: a review. J Investig Allergol Clin Immunol. 2002;12(3):143–154.
- Mamtani M, Sharma M, Amin M, Amin A, Jawahirani A, Kulkarni H. Erythrophagocytosis in sickle cell anemia: statistical evidence for a biological phenomenon. Med Hypotheses. 2007;68(5):1065–1070.
- Roitt IM, Male DK, Brostoff J. Immunology, 4th ed. London: Mosby, 1996.
- Booth C, Inusa B, Obaro SK. Infection in sickle cell disease: a review. Int J Infect Dis. 2010;14(1):e2–e12.
- Humbert JR, Winsor EL, Githens JM, Schmitz JB. Neutrophil dysfunctions in sickle cell disease. Biomed Pharmacotherap. 1990;44(3):153158.
- Donadi EA, Falcão RP. Neutrophil chemotaxis in sickle cell anaemia, sickle cell β0 thalassaemia, and after splenectomy. J Clin Pathol. 1987;40(6):670–672.
- Lachant N, Oseas R. Vaso-occlusive crisis-associated neutrophil dysfunction in patients with sickle-cell disease. Am J Med Sci. 1987;294(4):253–257.
- Hebbel RP. Beyond hemoglobin polymerization: the red blood cell membrane and sickle disease pathophysiology. Blood. 1991;15;77(2):214–237.
- Haywood LJ, Miselman HJ, Francis RB Jr. Effect of normal and sickle erythrocyte on prostacyclins released by perfused human umbilical cord veins. Am Hematol. 1992;40(4):267–282.
- Wood KC, Granger DN. Sickle cell disease: role of reactive oxygen and nitrogen metabolites. Clin Exp Pharmacol Physiol. 2007;34(9)926–932.