Hb ananosa or α₂111(G18)Ala → Valβ (α2 Mutation) and Hb mulhacen or α₂123(H6)Ala → Serβ₂ (αl Mutation) are two silent stable variants detected by sequencing of amplified dna

E. G. Kazanetz Laboratory of Protein Chemistry, Department of Biochemistry and Molecular Biology Medical College of Georgia, Augusta, GA, 30912-2100, USA

J. Ye. Leonova Laboratory of Protein Chemistry, Department of Biochemistry and Molecular Biology Medical College of Georgia, Augusta, GA, 30912-2100, USA

J. B. Wilson Laboratory of Protein Chemistry, Department of Biochemistry and Molecular Biology Medical College of Georgia, Augusta, GA, 30912-2100, USA

S. K. McMillan University Hygienic Laboratory, Des Moines Division Des, Moines, IA, 50319, USA

M. Walbrecht University Hygienic Laboratory, Des Moines Division Des, Moines, IA, 50319, USA

J. Mi. de Pablos Gallego Servicio de Hematologia, Hospital “Virgen de las Nieves” 18014, Granada, Spain

T. H. J. Huisman Laboratory of Protein Chemistry, Department of Biochemistry and Molecular Biology Medical College of Georgia, Augusta, GA, 30912-2100, USA

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Hb ananosa or α₂111(G18)Ala → Valβ (α2 Mutation) and Hb mulhacen or α₂123(H6)Ala → Serβ₂ (αl Mutation) are two silent stable variants detected by sequencing of amplified dna

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Hb ananosa or α2111(G18)Ala → Valβ (α2 Mutation) and Hb mulhacen or α2123(H6)Ala → Serβ2 (αl Mutation) are two silent stable variants detected by sequencing of amplified dna

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Hb ananosa or α₂111(G18)Ala → Valβ (α2 Mutation) and Hb mulhacen or α₂123(H6)Ala → Serβ₂ (αl Mutation) are two silent stable variants detected by sequencing of amplified dna