References
- Bellomo R, Wan L, Langenberg C, et al. Septic acute kidney injury: New concepts. Nephron Exp Nephrol. 2008;109:e95–e100
- Wang H, Bloom O, Zhang M, et al. HMG-1 as a late mediator of endotoxin lethality in mice. Science. 1999;285:248–251
- Asea A, Kraeft SK, Kurt-Jones EA, et al. HSP70 stimulates cytokine production through a CD14-dependant pathway, demonstrating its dual role as a chaperone and cytokine. Nat Med. 2000;6:435–442
- Thakar CV, Christianson A, Freyberg R, et al. Incidence and outcomes of acute kidney injury in intensive care units: A veterans administration study. Crit Care Med. 2009;37:2552–2558
- Wan L, Bagshaw SM, Langenberg C, et al. Pathophysiology of septic acute kidney injury: What do we really know? Crit Care Med. 2008;36:S198–S203
- Arslan F, Keogh B, McGuirk P, et al. TLR2 and TLR4 in ischemia reperfusion injury. Mediators Inflamm. 2010;2010:704202
- Gluba A, Banach M, Hannam S, et al. The role of Toll-like receptors in renal diseases. Nat Rev Nephrol. 2010;6:224–235
- Alves-Filho JC, Freitas A, Souto FO, et al. Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proc Natl Acad Sci USA. 2009;106:4018–4023
- Liang MD, Bagchi A, Warren HS, et al. Bacterial peptidoglycan-associated lipoprotein: A naturally occurring toll-like receptor 2 agonist that is shed into serum and has synergy with lipopolysaccharide. J Infect Dis. 2005;191:939–948
- Zahringer U, Lindner B, Inamura S, et al. TLR2 – promiscuous or specific? A critical re-evaluation of a receptor expressing apparent broad specificity. Immunobiology. 2008;213:205–224
- Oliveira-Nascimento L, Massari P, Wetzler LM. The role of TLR2 in infection and immunity. Front Immunol. 2012;3:79
- Knotek M, Rogachev B, Wang W, et al. Endotoxemic renal failure in mice: Role of tumor necrosis factor independent of inducible nitric oxide synthase. Kidney Int. 2001;59:2243–2249
- Castoldi A, Braga TT, Correa-Costa M, et al. TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis. PLoS One. 2012;7:e37584
- Lorenz E, Chemotti DC, Vandal K, et al. Toll-like receptor 2 represses nonpilus adhesin-induced signaling in acute infections with the Pseudomonas aeruginosa pilA mutant. Infect Immun. 2004;72:4561–4569
- Messaris E, Memos N, Chatzigianni E, et al. Apoptotic death of renal tubular cells in experimental sepsis. Surg Infect (Larchmt). 2008;9:377–388
- Schrier RW, Wang W. Acute renal failure and sepsis. N Engl J Med. 2004;351:159–169
- Armstrong L, Medford AR, Hunter KJ, et al. Differential expression of Toll-like receptor (TLR)-2 and TLR-4 on monocytes in human sepsis. Clin Exp Immunol. 2004;136:312–319
- Wolfs TG, Buurman WA, van Schadewijk A, et al. In vivo expression of Toll-like receptor 2 and 4 by renal epithelial cells: IFN-gamma and TNF-alpha mediated up-regulation during inflammation. J Immunol. 2002;168:1286–1293
- Cunningham PN, Wang Y, Guo R, et al. Role of Toll-like receptor 4 in endotoxin-induced acute renal failure. J Immunol. 2004;172:2629–2635
- Tiwari MM, Brock RW, Megyesi JK, et al. Disruption of renal peritubular blood flow in lipopolysaccharide-induced renal failure: Role of nitric oxide and caspases. Am J Physiol Renal Physiol. 2005;289:F1324–F1332
- Xu C, Chang A, Hack BK, et al. TNF-mediated damage to glomerular endothelium is an important determinant of acute kidney injury in sepsis. Kidney Int. 2014;85:72–81
- Pawar RD, Castrezana-Lopez L, Allam R, et al. Bacterial lipopeptide triggers massive albuminuria in murine lupus nephritis by activating Toll-like receptor 2 at the glomerular filtration barrier. Immunology. 2009;128:e206–e221
- Hotchkiss RS, Chang KC, Swanson PE, et al. Caspase inhibitors improve survival in sepsis: A critical role of the lymphocyte. Nat Immunol. 2000;1:496–501
- Oberholzer C, Oberholzer A, Clare-Salzler M, et al. Apoptosis in sepsis: A new target for therapeutic exploration. FASEB J. 2001;15:879–892
- Hotchkiss RS, Tinsley KW, Swanson PE, et al. Prevention of lymphocyte cell death in sepsis improves survival in mice. Proc Natl Acad Sci USA. 1999;96:14541–14546