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Original Article

Surgical Model of Volume Overload-Induced Ventricular Myocardial Hypertrophy (VHvo) to Study a Clinical Problem in Humans

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Pages 333-338 | Published online: 09 Jul 2009

References

  • Alyono D, Anderson R W., Pamsh D G., Dai X-Z, Bache R J. Alterations of myocardial blood flow associated with experimental canine left ventricular hypertrophy secondary to valvular aortic stenosis. Circ Rex 1966; 58(1)47–57
  • Olivetti G, Ricci R, Lagrasta C, Maniga E, Sonnenblick E H., Anversa P. Cellular basis of wall remodeling in long-term pressure overload-induced right ventricular hypertrophy in rats. Circ Res. 1988; 63(3)648–657
  • Cunningham M J., Apstein C S., Weinberg E O., Vogel W M., Lorell B H. Influence of glucose and insulin on the exaggerated diastolic and systolic dysfunction of hypertrophied rat hearts during hypoxia. Circ Res. 1990; 66(2)406–415
  • Knufman N MJ, van der Laarse A, Vliegen H W., Brinkman C JJ. Quantification of myocardial necrosis and cardiac hypertrophy in isoproterenol-treated rats. Chem Pathol Pharm. 1987; 57(1)15–32
  • Genovese A, De Alfieri W, Chiarello M, Latte S, Polverino W, Condorelli M. Development of cardiac hypertrophy in rats exposed to acute hypobaric hypoxia: studies with protein synthesis inhibitors. Exp Pathol. 1981; 20: 239–242
  • Isoyama S, Ito N, Kuroha M, Takishima T. Complete reversibility of physiological coronary vascular abnormalities in hypertrophied hearts produced by pressure overload in the rat. J Clin Invest. 1989; 84: 288–294
  • Litwin S E., Raya T E., Gay R G., et al. Chronic inhibition of fatty acid oxidation: new model of diastolic dysfunction. Am J Physiol. 1990; 258: H51–H56
  • Sin F M. Sympathetic changes during development of cardiac hypertrophy in aortic-constricted rats. Am J Physiol. 1988; 255: H452–H457
  • Sin F M., Nordin C, Factor S M., Sonnenblick E, Aronson R. Compensatory hypertrophy and failure in gradual pressure-overloaded guinea pig heart. Am J Physiol. 1989; 257: H1016–H1024
  • Vatner S F., Shannon R, Hittinger L. Reduced subendocardial coronary reserve: a potential mechanism for impaired diastolic function in the hypertrophied and failing heart. Circulation. 1990; 81(2, suppl III)III8–III14
  • Mirsky I, Laks M M. Time course of changes in the mechanical properties of the canine right and left ventricles during hypertrophy caused by pressure overload. Circ Res. 1980; 46(4)530–542
  • Attanan D E., Jones R N., Cume W D., et al. Characteristics of chronic left ventricular hypertrophy induced by subcoronary valvular aortic stenosis. J Thorac Cardiovasc Surg. 1981; 81: 382–388
  • Gaasch W H., Zile M R., Hoshino P K., Apstein C S., Blaustein A S. Stress-shortening relations and myocardial blood flow in compensated and failing canine hearts with pressure-overload hypertrophy. Circulation. 1989; 79(4)872–883
  • Kozlovskis P L., Fieber L A., Pruitt D K., et al. Myocardial changes during the progression of left ventricular pressure-overload by renal hypertension or aortic constriction: myosin, myosin ATPase and collagen. J Mol Cell Cardiol. 1987; 19: 105–114
  • Cooper G, IV, Tomanek R J., Ehrhardt J C., Marcus M L. Chronic progressive pressure overload of the cat right ventricle. Circ Res. 1981; 48(4)488–497
  • Harrison D G., Florentine M S., Brooks L A., Cooper S M., Marcus M L. The effect of hypertension and left ventricular hypertrophy on the lower range of coronary autoregulation. Circulation. 1988; 77(5)1108–1115
  • Legault F, Rouleau J L., Juneau C, Rose C, Rakusan K. Functional and morphological characteristics of compensated and decompensated cardiac hypertrophy in dogs with chronic infrarenal aorto-caval fistulas. Circ Res. 1990; 66(3)846–859
  • Liu S-K, Tilley L P. Animal models of primary myocardial diseases. Yale J Biol Med. 1980; 53: 191–211
  • Badke F R., White F C., Le Winter M, Covell J, Andres J, Bloor C. Effects of experimental volume-overload hypertrophy on myocardial blood flow and cardiac function. Am J Physiol. 1981; 241: H564–H570
  • Glauser E M. Advantages of piglets as experimental animals in pediatric research. Exp Med Surg. 1966; 24: 181–190

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