References
- De Leener A, Montanelli L and Van Durme Jet al. Presence and absence of follicle-stimulating hormone receptor mutations provide some insights into spontaneous ovarian hyperstimulation syndrome physiopathology. J Clin Endocrinol Metab 2006;91:555–62
- Aramwit P, Pruksananonda K, Kasettratat N and Jammeechai K. Risk factors for ovarian hyperstimulation syndrome in Thai patients using gonadotropins for in vitro fertilization. Am J Health Syst Pharm 2008;65:1148–53
- Wang TH, Horng SG and Chang CLet al. Human chorionic gonadotropin-induced ovarian hyperstimulation syndrome is associated with up-regulation of vascular endothelial growth factor. J Clin Endocrinol Metab 2002;87:3300–8
- Di Carlo C, Bruno P and Cirillo Det al. Increased concentrations of renin, aldosterone and Ca125 in a case of spontaneous, recurrent, familial, severe ovarian hyperstimulation syndrome. Hum Reprod 1997;12:2115–17
- Montanelli L, Delbaere A and Di Carlo Cet al. A mutation in the follicle-stimulating hormone receptor as a cause of familial spontaneous ovarian hyperstimulation syndrome. J Clin Endocrinol Metab 2004;89:1255–8
- Di Carlo C, Savoia F and Ferrara Cet al. Case report: a most peculiar family with spontaneous, recurrent ovarian hyperstimulation syndrome. Gynecol Endocrinol 2012;28:649–51
- Di Carlo C, Savoia F and Gargano Vet al. Successful pregnancy complicated by spontaneous, familial, recurrent ovarian hyperstimulation syndrome: report of two cases. Gynecol Endocrinol 2013;29:897–900
- Smits G, Campillo M and Govaerts Cet al. Glycoprotein hormone receptors: determinants in leucine-rich repeats responsible for ligand specificity. EMBO J 2003;22:2692–703
- Schubert RL, Narayan P and Puett D. Specificity of cognate ligand-receptor interactions: fusion proteins of human chorionic gonadotropin and the heptahelical receptors for human luteinizing hormone, thyroid-stimulating hormone, and follicle-stimulating hormone. Endocrinology 2003;144:129–37
- Kleinau G and Krause G. Thyrotropin and homologous glycoprotein hormone receptors: structural and functional aspects of extracellular signaling mechanisms. Endocr Rev 2009;30:133–51
- Anasti JN, Flack MR and Froehlich Jet al. A potential novel mechanism for precocious puberty in juvenile hypothyroidism. J Clin Endocrinol Metab 1995;80:276–9
- Smits G, Olatunbosun O and Delbaere Aet al. Ovarian hyperstimulation syndrome due to a mutation in the follicle-stimulating hormone receptor. N Engl J Med 2003;349:760–6
- Montanelli L, Van Durme JJ and Smits Get al. Modulation of ligand selectivity associated with activation of the transmembrane region of the human follitropin receptor. MolEndocrinol 2004;18:2061–73 N Engl J Med 2003;349:753–9
- Vasseur C, Rodien P and Beau Iet al. A chorionic gonadotropin-sensitive mutation in the follicle–stimulating hormone receptor as a cause of familial gestational spontaneous ovarian hyperstimulation syndrome. N Engl J Med 2003;349:753–9
- De Leener A, Caltabiano G and Erkan Set al. Identification of the first germline mutation in the extracellular domain of the follitropin receptor responsible for spontaneous ovarian hyperstimulation syndrome. Hum Mutat 2008;29:91–8
- Al Omari W, Ghazal-Aswad S, Sidky IH and Al Bassam MK. Ovarian salvage in bilaterally complicated severe ovarian hyperstimulation syndrome. Fertil Steril 2011;96:e77–9
- Rackow BW and Patrizio P. Successful pregnancy complicated by early and late adnexal torsion after in vitro fertilization. Fertil Steril 2007;87:697.e9–12
- Nandi-Munshi D, Tridgell A and Taplin CE. Acute ovarian torsion and primary hypothyroidism. Pediatrics 2013;132:e233–8