27
Views
7
CrossRef citations to date
0
Altmetric
Original Article

The Investigation of Karyotypic Instability in the High-Hyperdiploidy Subgroup of Acute Lymphoblastic Leukemia

, , &
Pages 187-193 | Published online: 01 Jul 2009

References

  • Stiller C A, Allen M B, Eatock E M. Childhood cancer in Britain: the National Registry of Childhood Tumours and incidence rates 1978–1987. Eur J Cancer 1995; 31A: 2028–2034
  • Grovas A, Fremgen A, Rauck A, Ruymann F B, Hutchinson C L, Winchester D P, Menck H R. The National Cancer Data Base report on patterns of childhood cancers in the United States. Cancer 1997; 80: 2321–32
  • Chessels J M, Swansbury G J, Reeves B, Bailey C C, Richards S M. Cytogenetics and prognosis in childhood lymphoblastic leukaemia: results of MRC UKALL X. Medical Research Council Working Party in Childhood Leukaemia. Br J Haematol 1997; 99: 93–100
  • Raimondi S C. Current status of cytogenetic research in childhood acute lymphoblastic leukemia. Blood 1993; 81: 2237–2251
  • Mertens F, Johansson B, Mitelman F. Dichotomy of hyperdiploid acute lymphoblastic leukemia on the basis of the distribution of gained chromosomes. Cancer Genet Cytogenet 1996; 92: 8–10
  • Moorman A V, Clark R, Farrell D M, Hawkins J M, Martineau M, Seeker-Walker L M. Probes for hidden hyperdiploidy in acute lymphoblastic leukaemia. Genes Chrom Cancer 1996; 16: 40–45
  • Raimondi S C, Pui C H, Hancock M L, Behm F G, Filatov L, Rivera G K. Heterogeneity of hyperdiploid (51–67) childhood acute lymphoblastic leukemia. Leukemia 1996; 10: 213–224
  • Mitelman F. ISCN (1995): An International System for Human Cytogenetic Nomenclature. S. Karger, Basel 1995
  • Onodera N, McCabe N R, Rubin C M. Formation of a hyperdiploid karyotype in childhood acute lymphoblastic leukemia. Blood 1992; 80: 203–8
  • Berger A, Strehl S, Hekele A, Ambros P F, Haas O A, Gadner H. Interphase cytogenetic study of childhood acute lymphoblastic leukemia. Med Pediatr Oncol 1994; 23: 413–421
  • Martin P L, Look A T, Schnell S, Harris M B, Pullen J, Shuster J J, Carroll A J, Pettenati M J, Nagesh Rao P. Comparison of fluorescence in situ hybridization, cytogenetic analysis, and DNA index analysis to detect chromosomes 4 and 10 aneuploidy in pediatric acute lymphoblastic leukemia: A Pediatric Oncology Group study. J Pediatr Hematol Oncol 1996; 18: 113–121
  • Ritterbach J, Hiddemann W, Beck J D, Schrappe M, Janka-Schaub G, Ludwig W-D, Harbott J, Lampert F. Detection of hyperdiploid leukemia (ALL) using fluorescence in situ hybridisation (FISH). Leukemia 1998; 12: 427–433
  • Small B E, Wood M, McConnell T S, Winter S S. Fluorescence in situ hybridization (FISH) detects clonal instability in anoptic nerve relapse of acute lymphoblastic leukemia. J Pediatr Hematol Oncol 1998; 20: 79–82
  • Seeker-Walker L M, Lawler S D, Hardisty R M. Prognostic implications of chromosomal findings in acute lymphoblastic leukaemia at diagnosis. BMJ 1978; 2: 1529–1530
  • Williams D L, Tsiatis A, Brodeur G M, Look A T, Melvin S L, Bowman W P, Kalwinsky D K, Rivera G, Dahl G V. Prognostic importance of chromosome number in 136 untreated children with acute lymphoblastic leukemia. Blood 1982; 60: 864–871
  • Raspers G J, Smets L A, Pieters R, van Zantwijk C H, van Wering E R, Veerman A J. Favorable prognosis of hyperdiploid common acute lymphoblastic leukemia may be explained by sensitivity to antimetabolites and other drugs: results of an in vitro study. Blood 1995; 85: 751–756
  • Whitehead V M, Vuchich M J, Cooley L D, Lauer S J, Mahoney D H, Shuster J J, Payment C, Koch P A, Akabutu J J, Bowen T, Kamen B A, Ravindranath Y, Emami A, Look A T, Beardsley G P, Pullen D J, Camitta B. Accumulation of methotrexate polyglutamates, ploidy and trisomies of both chromosomes 4 and 10 in lymphoblasts from children with B-progenitor cell acute lymphoblastic leukemia: a Pediatric Oncology Group Study. Leukemia Lymphoma 1998; 31: 507–519
  • Ito C, Kumagai M, Manabe A, Coustan-Smith E, Raimondi S C, Behm F G, Murti K G, Rubnitz J E, Pui C H, Campana D. Hyperdiploid acute lymphoblastic leukemia with 51 to 65 chromosomes: a distinct biological entity with a marked propensity to undergo apoptosis. Blood 1999; 93: 315–20

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.