REFERENCES
- Anonymous. Report of the National High Blood Pressure Education Program Working Group on high blood pressure in pregnancy. Am J Obstet Gynecol 2000; 183(1):S1–S22.
- Maynard SE, Min J-Y, Merchan J, . Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. J Clin Invest 2003; 111(5):649–658.
- Venkatesha S, Toporsian M, Lam C, . Soluble endoglin contributes to the pathogenesis of preeclampsia. Nat Med 2006; 12(6):642–649.
- Noori M, Donald AE, Angelakopoulou A, Hingorani AD, Williams DJ. Prospective study of placental angiogenic factors and maternal vascular function before and after preeclampsia and gestational hypertension. Circulation 2010; 122(5):478–487.
- Ahmad S, Ahmed A. Elevated placental soluble vascular endothelial growth factor receptor-1 inhibits angiogenesis in preeclampsia. Circ Res 2004; 95(9):884–891.
- Machnik A, Neuhofer W, Jantsch J, . Macrophages regulate salt-dependent volume and blood pressure by a vascular endothelial growth factor-C-dependent buffering mechanism. Nat Med 2009; 15(5):545–552.
- Karumanchi SA, Maynard SE, Stillman IE, Epstein FH, Sukhatme VP. Preeclampsia: A renal perspective. Kidney Int 2005; 67(6):2101–2113.
- Strauss MB. Observations on the etiology of the toxemias of pregnancy. II. Production of acute exacerbation of toxemia by sodium salts in pregnant women with hypoproteinemia. Am J Med Sci 1937; 194(6):185–199.
- Gallery ED, Brown MA. Control of sodium excretion in human pregnancy. Am J Kidney Dis 1987; 9(4):290–295.
- Roberts JM, Cooper DW. Pathogenesis and genetics of pre-eclampsia. Lancet 2001; 357(9249):53–56.
- Dunk C, Ahmed A. Expression of VEGF-C and activation of its receptors sVEGFR-2 and VEGFR-3 in trophoblast. Histol Histopathol 2001; 16(2):359–375.
- Chung J-Y, Song Y, Wang Y, Magness RR, Zheng J. Differential expression of vascular endothelial growth factor (VEGF), endocrine gland derived-VEGF, and VEGF receptors in human placentas from normal and preeclamptic pregnancies. J Clin Endocrinol Metab 2004; 89(5): 2484–2490.
- Zhou Y, McMaster M, Woo K, . Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets syndrome. Am J Pathol 2002; 160(4): 1405–1423.
- Machnik A, Dahlmann A, Kopp C, . Mononuclear phagocyte system depletion blocks interstitial tonicity-responsive enhancer binding protein/vascular endothelial growth factor C expression and induces salt-sensitive hypertension in rats. Hypertension 2010; 55(3):755–761.
- Slagman MCJ, Kwakernaak AJ, Yazdani S, . Vascular endothelial growth factor C levels are modulated by dietary salt intake in proteinuric chronic kidney disease patients and in healthy subjects. Nephrol Dial Transplant 2012; 27(3):978–982.
- Levine RJ, Maynard SE, Qian C, . Circulating angiogenic factors and the risk of preeclampsia. N Engl J Med 2004; 350(7):672–683.
- Rajakumar A, Cerdeira AS, Rana S, . Transcriptionally active syncytial aggregates in the maternal circulation may contribute to circulating soluble fms-like tyrosine kinase 1 in preeclampsia. Hypertension 2012; 59(2):256–264.
- Bujold E, Romero R, Chaiworapongsa T, . Evidence supporting that the excess of the sVEGFR-1 concentration in maternal plasma in preeclampsia has a uterine origin. J Matern Fetal Neonatal Med 2005; 18(1):9–16.