References
- American College of Chest Physicians/Society of Critical Care Medicine Consensus Conference: definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. (1992). Crit Care Med 20:864–74.
- Belge KU, Dayyani F, Horelt A, et al. (2002). The proinflammatory CD14+CD16+DR++ monocytes are a major source of TNF. J Immunol 168:3536–42.
- Dellinger RP, Levy MM, Rhodes A, et al. (2013). Surviving sepsis campaign: international guidelines for management of severe sepsis and septic shock: 2012. Crit Care Med 41:580–637.
- Fingerle G, Pforte A, Passlick B, et al. (1993). The novel subset of CD14+/CD16+ blood monocytes is expanded in sepsis patients. Blood 82:3170–6.
- Furman MI, Barnard MR, Krueger LA, et al. (2001). Circulating monocyte-platelet aggregates are an early marker of acute myocardial infarction. J Am Coll Cardiol 38:1002–6.
- Guerin E, Orabona M, Raquil MA, et al. (2014). Circulating immature granulocytes with T-cell killing functions predict sepsis deterioration. Crit Care Med 42:2007–18.
- Hilgers KF. (2002). Monocytes/macrophages in hypertension. J Hypertens 20:593–6.
- Kaukonen KM, Bailey M, Suzuki S, et al. (2014). Mortality related to severe sepsis and septic shock among critically ill patients in Australia and New Zealand, 2000–2012. JAMA 311:1308.
- Munoz C, Carlet J, Fitting C, et al. (1991). Dysregulation of in vitro cytokine production by monocytes during sepsis. J Clin Invest 88:1747–54.
- Passlick B, Flieger D, Ziegler-Heitbrock HW. (1989). Identification and characterization of a novel monocyte subpopulation in human peripheral blood. Blood 74:2527–34.
- Redondo AC, Ceccon ME, Silveira-Lessa AL, et al. (2014). TLR-2 and TLR-4 expression in monocytes of newborns with late-onset sepsis. J Pediatr 90:472–8.
- Rogacev KS, Seiler S, Zawada AM, et al. (2011). CD14++CD16+ monocytes and cardiovascular outcome in patients with chronic kidney disease. Eur Heart J 32:84–92.
- Rogacev KS, Ulrich C, Blomer L, et al. (2010). Monocyte heterogeneity in obesity and subclinical atherosclerosis. Eur Heart J 31:369–76.
- Rondina MT, Carlisle M, Fraughton T, et al. (2015). Platelet-monocyte aggregate formation and mortality risk in older patients with severe sepsis and septic shock. J Gerontol A Biol Sci Med Sci 70:225–31.
- Rondina MT, Grissom CK, Men S, et al. (2012). Whole blood flow cytometry measurements of in vivo platelet activation in critically-Ill patients are influenced by variability in blood sampling techniques. Thromb Res 129:729–35.
- Semple JW, Freedman J. (2010). Platelets and innate immunity. Cell Mol Life Sci 67:499–511.
- Sener A, Ozsavci D, Oba R, et al. (2005). Do platelet apoptosis, activation, aggregation, lipid peroxidation and platelet-leukocyte aggregate formation occur simultaneously in hyperlipidemia? Clin Biochem 38:1081–7.
- Shantsila E, Lip GY. (2009). Monocytes in acute coronary syndromes. Arterioscler Thromb Vasc Biol 29:1433–8.
- Shantsila E, Lip GY. (2009). The role of monocytes in thrombotic disorders. Insights from tissue factor, monocyte-platelet aggregates and novel mechanisms. Thromb Haemost 102:916–24.
- Tapp LD, Shantsila E, Wrigley BJ, et al. (2012). The CD14++CD16+ monocyte subset and monocyte-platelet interactions in patients with ST-elevation myocardial infarction. J Thromb Haemost 10:1231–41.
- Vandendries ER, Furie BC, Furie B. (2004). Role of P-selectin and PSGL-1 in coagulation and thrombosis. Thromb Haemost 92:459–66.
- Wong KL, Tai JJ, Wong WC, et al. (2011). Gene expression profiling reveals the defining features of the classical, intermediate, and nonclassical human monocyte subsets. Blood 118:e16–31.
- Wu Q, Hu D, Ren J. (2015). Chronic critical illness should be considered in long-term mortality study among critical illness patients. Crit Care Med 43:e57.
- Wu Q, Ren J, Hu D, et al. (2015). An elevated percentage of reticulated platelet is associated with increased mortality in septic shock patients. Medicine (Baltimore) 94:e814.
- Zawada AM, Rogacev KS, Rotter B, et al. (2011). SuperSAGE evidence for CD14++CD16+ monocytes as a third monocyte subset. Blood 118:e50–61.
- Zhou X, Zhang L, Ji WJ, et al. (2013). Variation in dietary salt intake induces coordinated dynamics of monocyte subsets and monocyte-platelet aggregates in humans: implications in end organ inflammation. PLoS One 8:e60332.
- Ziegler-Heitbrock L, Ancuta P, Crowe S, et al. (2010). Nomenclature of monocytes and dendritic cells in blood. Blood 116:e74–80.
- Ziegler-Heitbrock L. (2007). The CD14+ CD16+ blood monocytes: their role in infection and inflammation. J Leukoc Biol 81:584–92.
- Ziegler-Heitbrock L. (2014). Monocyte subsets in man and other species. Cell Immunol 289:135–9.