References
- Brain Energy Metabolism, B. K. Siesjö. John Wiley, Chichester 1978
- Siesjö B. K. Cell damage in the brain: A speculative synthesis. J Cereb Blood Flow Metabol 1981; 1: 155–185
- Hossmann K.-A. Treatment of experimental cerebral ischemia. J Cereb Blood Flow Metabol 1982; 2: 275–297
- Kågström E., Smith M.-L., Siesjö B. K. Local cerebral blood flow in the recovery period following complete cerebral ischemia in the rat. J Cereb Blood Flow Metabol 1983; 2: 183–192
- Brierley J. B. Cerebral hypoxia. Greenf ie Iďs Neuropathology, W. Blackwood, J. A.N. Corsellis. Edward Arnold, London 1976
- Kirino T. Delayed neuronal death in the gerbil hippocampus following ischemia. Brain Res 1982; 239: 57–69
- Smith M.-L., Auer R. N., Siesjö B. K. The density and distribution of ischemic brain injury in the rat following 2–10 min of forebrain ischemia. Acta Neuropathol (Berl) 1984; 64: 319–332
- Hossmann K.-A., Kleihues P. Reversibility of ischemia brain damage. Arch Neurol 1973; 22: 375–384
- Ito U., Spatz M., Walker J. T., Jr., Klatzo I. Experimental cerebral ischemia inmongolian gerbils. I. Light microscopic observations. Acta Neuropathol (Berl) 1975; 32: 209–223
- Ito U., Go K. G., Walker J. T., Jr., Spatz M., Klatzo I. Experimental cerebral ischemia in mongolian gerbil s. III. Behaviour of the blood-brain barrier. Acta Neuropathol (Berl) 1976; 34: 1–6
- Suzuki R., Yamaguchi T., Choh-Luh L., Klatzo I. The effects of 5-minute ischemia in mongolian gerbils. II. Changes of spontaneous neuronal activity in cerebral cortex and CA1 sector of hippocampus. Acta Neuropathol (Berl) 1983; 60: 217–222
- Pulsinelli W. A., Waldman S., Rawlinson D., Plum F. Hyperglycemia converts ischemic neuronal damage into brain infarction. Neurology (NY) 1982; 32: 1239–1246
- Pulsinelli W. A., Duffy T. E. Regional energy balance in rat brain after transient forebrain ischemia. J. Neurochem 1983; 40: 1500–1503
- Auer R. N., Olsson Y., Siesjö B. K. Hypoglycemic brain injury in the rat. Correlation of density of brain damage with the EEG isoelectric time: A quantitative study. Diabetes 1984; 33: 1090–1098
- Auer R. N., Wieloch T., Olsson Y., Siesjö B. K. The distribution of hypoglycemia brain damage. Acta Neuropathol (Berl) 1984; 64: 177–191
- Nevander G., Ingvar M., Auer R., Siesjö B. K. Irreversible neuronal damage after short periods of status epilepticus. Acta Physiol Scand 1984; 120: 155–157
- Nevander G., Ingvar M., Auer R., Siesjö B. K. Status epilepticus in well oxygenated rats causes neuronal necrosis. Ann Neurol 1985, in press
- Wieloch T., Siesjö B. K. Ischemic brain injury: The importance of calcium lipolytic activities. and free fatty acids, Path Biol 1982; 5: 269–277
- Siesjö B. K., Wieloch T. Fatty acid metabolism and the mechanisms of ischemic brain damage. Cerebrovascular Diseases, M. Reivich, H. I. Hurtig. Raven Press, New York 1983; 251–268
- Siesjö B. K., Wieloch T. Cerebral metabolism in ischemia: neurochemical basis for therapy. Br J Anaesthesia 1985; 57, in press
- Siesjö B. K., Wieloch T. Brain injury: Neurochemical aspects. Central Nervous System Trauma, J. T. Povlishock, D. P. Becker. William Byrd Press, Richmond, VA 1985, in press
- Myers R. E. Lactic acid accumulation as cause of brain edema and cerebral necrosis resulting from oxygen deprivation. Advances in Perinatal Neurology, R. Korobkin, G. Guilleminault. Spectrum, New York 1979; 85–114
- Plum F. What causes infarction in ischemic brain?: The Robert Wartenberg Lecture. Neurology 1983; 33: 222–233
- Siesjö B. K. Cerebral circulation and metabolism. J Neurosurg 1984; 60: 883–908
- Siesjö B. K. Acid-base homeostasis in the brain: physiology, chemistry, and neurochemical pathology. Progress in Brain Res. 1985, in press
- Hillered L., Smith M.-L., Siesjö B. K. Lactic acidosis and recovery of mitochondrial function following forebrain ischemia in the rat. J Cereb Blood Flow Metabol 1985; 5, in press
- Lynch G., Baudry M. The biochemistry of memory: A new and specific hypothesis. Science 1984; 224: 1057–1063
- Rasmussen H., Waisman D. M. Modulation of cell function in the calcium messenger system. Rev Physiol Biochem Pharmacol. 1983; 21: 111–148