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Modern Rheumatology Volume 18, 2008 - Issue 4
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Letter

HLA-B27-transgenic rats, amyloid deposits, and spondyloarthropathies

Barbara Uchanska-ZieglerInstitut für Immungenetik, Charité-Universitätsmedizin Berlin, Campus Benjamin Franklin, Freie Universität Berlin, Thielallee 73, Berlin 14195, GermanyCorrespondence[email protected]
&
Andreas ZieglerInstitut für Immungenetik, Charité-Universitätsmedizin Berlin, Campus Benjamin Franklin, Freie Universität Berlin, Thielallee 73, Berlin 14195, Germany
Pages 425-426 | Received 18 Feb 2008, Accepted 05 Mar 2008, Published online: 02 Jan 2014

References

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  • Tran TM, Dorris ML, Satumtira N, Richardson JA, Hammer RE, Shang J, et al. Additional human beta2 m curbs HLA-B27 mis-folding and promotes arthritis and spondylitis without colitis in male HLA-B27 transgenic rats. Arthritis Rheum. 2006;54:1317–27.
  • Ziegler A, Loll B, Misselwitz R, Uchanska-Ziegler B. Implica-tions of structural and thermodynamic studies of HLA-B27 subtypes exhibiting differential association with ankylosing spondylitis. In: López-Larrea C, editor. Molecular mechanisms of spondyloarthropathies. Austin: Landes Bioscience; 2008 (in press).
  • Uchanska-Ziegler B, Ziegler A. Ankylosing spondylitis: a fign-deposition disease? Trends Immunol. 2003;24:73–6.

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