Alterations in B cell development, CDR-H3 repertoire and dsDNA-binding antibody production among C57BL/6 ΔD−iD mice congenic for the lupus susceptibility loci sle1, sle2 or sle3

Mohamed KhassDepartment of Medicine, University of Alabama at Birmingham, Birmingham, AL, USA, ;Genetic Engineering and Biotechnology Division, National Research Center, Cairo, Egypt,

Robert L. SchelonkaDepartment of Pediatrics, University of Alabama at Birmingham, Birmingham, AL, USA,

Cun Ren LiuDepartment of Medicine, University of Alabama at Birmingham, Birmingham, AL, USA,

Ada ElgavishDepartment of Medicine, University of Alabama at Birmingham, Birmingham, AL, USA,

Laurence MorelDepartment of Pathology, University of Florida, Gainesville, FL, USA, and

Peter D. BurrowsDepartment of Microbiology, University of Alabama at Birmingham, Birmingham, AL, USA

Harry W. Schroeder JrDepartment of Medicine, University of Alabama at Birmingham, Birmingham, AL, USA, ;Department of Microbiology, University of Alabama at Birmingham, Birmingham, AL, USACorrespondence[email protected]

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Alterations in B cell development, CDR-H3 repertoire and dsDNA-binding antibody production among C57BL/6 ΔD−iD mice congenic for the lupus susceptibility loci sle1, sle2 or sle3

Absolute_B_cell_numbers_8-15-2016.pdf

Sle_DD-iD_sequence_analysis_8-15-2016.pdf

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Alterations in B cell development, CDR-H3 repertoire and dsDNA-binding antibody production among C57BL/6 ΔD−iD mice congenic for the lupus susceptibility loci sle1, sle2 or sle3

Absolute_B_cell_numbers_8-15-2016.pdf

Sle_DD-iD_sequence_analysis_8-15-2016.pdf

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