METTL3-mediated m⁶A modification of lnc KCNQ1OT1 promotes doxorubicin resistance in breast cancer by regulating miR-103a-3p/MDR1 axis

Zhiyang Zhoua Department of Breast Surgery, Xiangya Hospital, Central South University, Changsha, Hunan, China;b Clinical Research Center For Breast Cancer In Hunan Province, Changsha, Hunan, China;c National Clinical Research Center for Geriatric Disorders, Changsha, Hunan, ChinaView further author information

Yukun Caoa Department of Breast Surgery, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaView further author information

Yuan Yanga Department of Breast Surgery, Xiangya Hospital, Central South University, Changsha, Hunan, China;c National Clinical Research Center for Geriatric Disorders, Changsha, Hunan, ChinaView further author information

Shouman Wanga Department of Breast Surgery, Xiangya Hospital, Central South University, Changsha, Hunan, China;b Clinical Research Center For Breast Cancer In Hunan Province, Changsha, Hunan, China;c National Clinical Research Center for Geriatric Disorders, Changsha, Hunan, ChinaView further author information

Feiyu Chena Department of Breast Surgery, Xiangya Hospital, Central South University, Changsha, Hunan, China;b Clinical Research Center For Breast Cancer In Hunan Province, Changsha, Hunan, China;c National Clinical Research Center for Geriatric Disorders, Changsha, Hunan, ChinaCorrespondence[email protected]
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METTL3-mediated m⁶A modification of lnc KCNQ1OT1 promotes doxorubicin resistance in breast cancer by regulating miR-103a-3p/MDR1 axis

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METTL3-mediated m6A modification of lnc KCNQ1OT1 promotes doxorubicin resistance in breast cancer by regulating miR-103a-3p/MDR1 axis

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METTL3-mediated m⁶A modification of lnc KCNQ1OT1 promotes doxorubicin resistance in breast cancer by regulating miR-103a-3p/MDR1 axis