Supplemental material
Cogent Biology
Volume 3, 2017 - Issue 1
Short Communication
The rough fur (ruf) mutation in mice is an allele of myelin protein zero-like 3 (Mpzl3)
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Thomas R. KingDepartment of Biomolecular Sciences, Central Connecticut State University, 1615 Stanley Street, New Britain, CT06053, USACorrespondence[email protected]
https://orcid.org/0000-0001-6332-5484View further author information
https://orcid.org/0000-0001-6332-5484View further author information |
(Reviewing Editor)
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Network Analysis Identifies Mitochondrial Regulation of Epidermal Differentiation by MPZL3 and FDXR.
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Seborrhea-like dermatitis with psoriasiform elements caused by a mutation in ZNF750, encoding a putative C2H2 zinc finger protein.
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Springer Science and Business Media LLC
Genetics analysis of mouse mutations Abnormal feet and tail and rough coat, which cause developmental abnormalities and alopecia.
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Springer Science and Business Media LLC
The rough fur (ruf) mutation in mice is an allele of myelin protein zero-like 3 (Mpzl3)
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Taylor & Francis
Mutation in Mpzl3, a Gene Encoding a Predicted the Adhesion Protein, in the Rough Coat (rc) Mice with Severe Skin and Hair Abnormalities
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Elsevier BV
The spontaneous juvenile alopecia (jal) mutation in mice is associated with the insertion of an IAP element in the Gata3 gene
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Informa UK Limited
ZNF750 interacts with KLF4 and RCOR1, KDM1A, and CTBP1/2 chromatin regulators to repress epidermal progenitor genes and induce differentiation genes
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Cold Spring Harbor Laboratory
Retroviral elements and their hosts: insertional mutagenesis in the mouse germ line.
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Public Library of Science (PLoS)
The juvenile alopecia mutation (jal) maps to mouse Chromosome 2, and is an allele of GATA binding protein 3 (Gata3).
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Springer Science and Business Media LLC
Genetic ablation of myelin protein zero-like 3 in mice increases energy expenditure, improves glycemic control, and reduces hepatic lipid synthesis.
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American Physiological Society
Mll Has a Critical Role in Fetal and Adult Hematopoietic Stem Cell Self-Renewal
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Elsevier Inc.
A mouse knockout library for secreted and transmembrane proteins.
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Springer Science and Business Media LLC
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