HIF-1α-independent hypoxia-induced rapid PTK6 stabilization is associated with increased motility and invasion

Isabel M Pires CR-UK/MRC Oxford Institute for Radiation Oncology; Department of Oncology; University of Oxford; Oxford, UK; School of Biological, Biomedical and Environmental Sciences; University of Hull; Hull, UKCorrespondence[email protected] [email protected]

Nina JG Blokland CR-UK/MRC Oxford Institute for Radiation Oncology; Department of Oncology; University of Oxford; Oxford, UK

Agnieke WT Broos CR-UK/MRC Oxford Institute for Radiation Oncology; Department of Oncology; University of Oxford; Oxford, UK

Flore-Anne Poujade School of Biological, Biomedical and Environmental Sciences; University of Hull; Hull, UK

Joana M Senra CR-UK/MRC Oxford Institute for Radiation Oncology; Department of Oncology; University of Oxford; Oxford, UK

Suzanne A Eccles Cancer Research UK Cancer Therapeutics Unit; The Institute of Cancer Research; Sutton, UK

Paul N Span Radboud University Nijmegen Medical Centre; Radiation Oncology; Nijmegen, the Netherlands

Amanda J Harvey Biosciences; Brunel Institute for Cancer Genetics and Pharmacogenomics; Brunel University; Uxbridge, UKCorrespondence[email protected] [email protected]

Ester M Hammond CR-UK/MRC Oxford Institute for Radiation Oncology; Department of Oncology; University of Oxford; Oxford, UK

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HIF-1α-independent hypoxia-induced rapid PTK6 stabilization is associated with increased motility and invasion

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HIF-1α-independent hypoxia-induced rapid PTK6 stabilization is associated with increased motility and invasion

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