Pannexin1 Single Nucleotide Polymorphism and Platelet Reactivity in a Cohort of Cardiovascular Patients

Florian B. StierlinDepartment of Pathology and Immunology, University of Geneva, Geneva, Switzerland; ;Department of Medical Specializations – Cardiology, University of Geneva, Geneva, Switzerland; ;Geneva Platelet Group, University of Geneva, Geneva, Switzerland; View further author information

Filippo MolicaDepartment of Pathology and Immunology, University of Geneva, Geneva, Switzerland; ;Department of Medical Specializations – Cardiology, University of Geneva, Geneva, Switzerland; View further author information

Jean-Luc RenyGeneva Platelet Group, University of Geneva, Geneva, Switzerland; ;Department of Medical Specializations, Division of Internal Medicine and Rehabilitation, Trois-Chêne Hospital, Geneva University Hospitals, Geneva, Switzerland;

http://orcid.org/0000-0002-7528-7363 View further author information

Brenda R. KwakDepartment of Pathology and Immunology, University of Geneva, Geneva, Switzerland; ;Department of Medical Specializations – Cardiology, University of Geneva, Geneva, Switzerland; Correspondence[email protected]

http://orcid.org/0000-0003-0015-0044 View further author information

Pierre FontanaGeneva Platelet Group, University of Geneva, Geneva, Switzerland; ;Department of Medical Specializations, Division of Angiology and Haemostasis, Geneva University Hospitals, Geneva, Switzerland

http://orcid.org/0000-0003-1546-0774 View further author information

Pannexin1 Single Nucleotide Polymorphism and Platelet Reactivity in a Cohort of Cardiovascular Patients

Table 1. Mean percentage of collagen induced-platelet aggregation according to Panx1-400A > C genotypes stratified by anti-platelet treatments.

Table 2. Association between recurrence of MACE in the ADRIE population and the Panx1-400A > C SNP.

Table 3. Panx1-400A > C SNP in 26 patients with extreme platelet reactivity (high or low PRI).

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Pannexin1 Single Nucleotide Polymorphism and Platelet Reactivity in a Cohort of Cardiovascular Patients

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Table 1. Mean percentage of collagen induced-platelet aggregation according to Panx1-400A > C genotypes stratified by anti-platelet treatments.

Table 2. Association between recurrence of MACE in the ADRIE population and the Panx1-400A > C SNP.

Table 3. Panx1-400A > C SNP in 26 patients with extreme platelet reactivity (high or low PRI).

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