Hb Zoetermeer: A New Mutation on the α2 Gene Inducing an Ala→Ser Substitution at Codon 21 is Possibly Associated with a Mild Thalassemic Phenotype

Cornelis L. Harteveld The Hemoglobinopathies Laboratory, Department of Human and Clinical Genetics, Leiden University Medical Center, Leiden, The Netherlands

Willem C.H. van Helden Department of Clinical Chemistry, Lange Land Hospital, Zoetermeer, The Netherlands

George L. Boxma General Practitioner, Zoetermeer, The Netherlands

Peter van Delft The Hemoglobinopathies Laboratory, Department of Human and Clinical Genetics, Leiden University Medical Center, Leiden, The Netherlands

Margaretha Bakker-Verweij The Hemoglobinopathies Laboratory, Department of Human and Clinical Genetics, Leiden University Medical Center, Leiden, The Netherlands

Henri Wajcman INSERM U654, Créteil, France; Laboratoire de Biochimie, Hôpital Henri Mondor, AP-HP, Créteil, France

Isabelle Zanella-Cleon Institut de Biologie et Chimie des Protéines (IFR128), Lyon, France

Michel Becchi Institut de Biologie et Chimie des Protéines (IFR128), Lyon, France

Piero C. Giordano The Hemoglobinopathies Laboratory, Department of Human and Clinical Genetics, Leiden University Medical Center, Leiden, The NetherlandsCorrespondence[email protected]

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Hb Zoetermeer: A New Mutation on the α2 Gene Inducing an Ala→Ser Substitution at Codon 21 is Possibly Associated with a Mild Thalassemic Phenotype

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Hb Zoetermeer: A New Mutation on the α2 Gene Inducing an Ala→Ser Substitution at Codon 21 is Possibly Associated with a Mild Thalassemic Phenotype

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