Inhibition of EGFR Signaling and Activation of Mitochondrial Apoptosis Contribute to Tanshinone IIA-Mediated Tumor Suppression in Non-Small Cell Lung Cancer Cells

Feng Gao1 Cell Transplantation and Gene Therapy Institute, The Third Xiangya Hospital, Central South University, Changsha, Hunan410013, People’s Republic of China;2 Department of Ultrasonography, The Third Xiangya Hospital of Central South University, Changsha, Hunan410013, People’s Republic of China

Ming Li1 Cell Transplantation and Gene Therapy Institute, The Third Xiangya Hospital, Central South University, Changsha, Hunan410013, People’s Republic of China;3 School of Stomatology, Hunan University of Chinese Medicine, Changsha, Hunan410208, People’s Republic of China;4 Changsha Stomatological Hospital, Hunan University of Chinese Medicine, Changsha, Hunan, 410004, People’s Republic of China

Wenbin Liu5 Department of Pathology, The Affiliated Hunan Cancer Hospital of Central South University, Changsha, Hunan410013, People’s Republic of China

Wei Li2 Department of Ultrasonography, The Third Xiangya Hospital of Central South University, Changsha, Hunan410013, People’s Republic of China;6 Department of Radiology, The Third Xiangya Hospital of Central South University, Changsha, Hunan410013, People’s Republic of ChinaCorrespondence[email protected]

https://orcid.org/0000-0002-7267-6494

Inhibition of EGFR Signaling and Activation of Mitochondrial Apoptosis Contribute to Tanshinone IIA-Mediated Tumor Suppression in Non-Small Cell Lung Cancer Cells

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Inhibition of EGFR Signaling and Activation of Mitochondrial Apoptosis Contribute to Tanshinone IIA-Mediated Tumor Suppression in Non-Small Cell Lung Cancer Cells

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