TAK1 promotes cell survival by TNFAIP3 and IL-8 dependent and NF-κB independent pathway in HeLa cells exposed to heat stress

Peng LiDepartment of Radiological Sciences, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Sugitani ToyamaJapan

Yukihiro FurusawaDepartment of Radiological Sciences, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Sugitani ToyamaJapan

Zheng-Li WeiDepartment of Radiological Sciences, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Sugitani ToyamaJapan

Hiroaki SakuraiDepartment of Cancer Cell Biology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama SugitaniToyama

Yoshiaki TabuchiDivision of Molecular Genetics Research, Life Science Research Center, University of Toyama SugitaniToyama

Qing-Li ZhaoDepartment of Radiological Sciences, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Sugitani ToyamaJapan

Ikuo SaikiDivision of Pathogenic Biochemistry, Institute of Natural Medicine, University of Toyama, Sugitani ToyamaJapan

Takashi KondoDepartment of Radiological Sciences, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, Sugitani ToyamaJapanCorrespondence[email protected]

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TAK1 promotes cell survival by TNFAIP3 and IL-8 dependent and NF-κB independent pathway in HeLa cells exposed to heat stress

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TAK1 promotes cell survival by TNFAIP3 and IL-8 dependent and NF-κB independent pathway in HeLa cells exposed to heat stress

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