α⁺-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup]

John S. WayeHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada, ;Department of Pathology and Molecular Medicine, McMaster University, Hamilton, Ontario, CanadaCorrespondence[email protected]

Barry EngHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada,

Meredith HannaHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada,

Betty-Ann HohenadelHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada,

Lisa M. NakamuraHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada,

Lynda WalkerHamilton Regional Laboratory Medicine Program, Hamilton Health Sciences, Hamilton, Ontario, Canada,

α⁺-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup]

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α+-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup]

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α⁺-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup]