Antioxidative effects of ethyl 2-(3-(benzo[d]thiazol-2-yl)ureido)acetate against amyloid β-induced oxidative cell death via NF-κB, GSK-3β and β-catenin signaling pathways in cultured cortical neurons

E.-A. KimDepartment of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul, Korea

C. H. ChoDepartment of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul, Korea

D. W. KimDepartment of Biochemistry and Molecular Biology, Research Institute of Oral Sciences, College of Dentistry, Kangnung-Wonju National University, Gangneung, Korea

S. Y. ChoiDepartment of Biomedical Science and Research Institute of Bioscience and Biotechnology, Hallym University, Chuncheon, Korea

J.-W. HuhDepartment of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul, KoreaCorrespondence[email protected] [email protected]

S.-W. ChoDepartment of Biochemistry and Molecular Biology, University of Ulsan College of Medicine, Seoul, KoreaCorrespondence[email protected] [email protected]

Antioxidative effects of ethyl 2-(3-(benzo[d]thiazol-2-yl)ureido)acetate against amyloid β-induced oxidative cell death via NF-κB, GSK-3β and β-catenin signaling pathways in cultured cortical neurons

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Antioxidative effects of ethyl 2-(3-(benzo[d]thiazol-2-yl)ureido)acetate against amyloid β-induced oxidative cell death via NF-κB, GSK-3β and β-catenin signaling pathways in cultured cortical neurons

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