References
- Wordsworth B. P., Bell J. I. The immunogenetics of rheumatoid arthritis. Springer Semin Immunopathol 1992; 14: 59–78
- MacGregor A. J., Snieder H., Rigby A. S., Koskenvuo M., Kaprio J., Aho K., et al. Characterizing the quantitative genetic contribution to rheumatoid arthritis using data from twins. Arthritis Rheum 2000; 43: 30–7
- Harney S. M., Newton J. L., Wordsworth B. P. Molecular genetics of rheumatoid arthritis. Curr Opin Pharmacol 2003; 3: 280–5
- Kinne R. W., Brauer R., Stuhlmuller B., Palombo‐Kinne E., Burmester G. R. Macrophages in rheumatoid arthritis. Arthritis Res 2000; 2: 189–202
- Wright S. D., Ramos R. A., Tobias P. S., Ulevitch R. J., Mathison J. C. CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein. Science 1990; 249: 1431–3
- Dobrovolskaia M. A., Vogel S. N. Toll receptors, CD14, and macrophage activation and deactivation by LPS. Microbes Infect 2002; 4: 903–14
- Baldini M., Lohman I. C., Halonen M., Erickson R. P., Holt P. G., Martinez F. D. A polymorphism in the 5′ flanking region of the CD14 gene is associated with circulating soluble CD14 levels and with total serum immunoglobulin E. Am J Respir Cell Mol Biol 1999; 20: 976–83
- Hubacek J. A., Rothe G., Pitha J., Skodova Z., Stanek V., Poledne R., et al. C(−260)→T polymorphism in the promoter of the CD14 monocyte receptor gene as a risk factor for myocardial infarction. Circulation 1999; 99: 3218–220
- Arnett F. C., Edworthy S. M., Bloch D. A., McShane D. J., Fries J. F., Cooper N. S., et al. The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis. Arthritis Rheum 1988; 31: 315–24
- Patino R., Ibarra J., Rodriguez A., Yague M. R., Pintor E., Fernandez‐Cruz A., et al. Circulating monocytes in patients with diabetes mellitus, arterial disease, and increased CD14 expression. Am J Cardiol 2000; 85: 1288–91
- Koenig W., Khuseyinova N., Hoffmann M. M., Marz W., Frohlich M., Hoffmeister A., et al. CD14 C(−260)→T polymorphism, plasma levels of the soluble endotoxin receptor CD14, their association with chronic infections and risk of stable coronary artery disease. J Am Coll Cardiol 2002; 40: 34–42
- Hubacek J. A., Stavek P., Pitha J., Skodova Z., Poledne R. CD14 (C−159→T) polymorphism and levels of C‐reactive protein. Cas Lek Cesk 2003; 142: 174–6
- LeVan T. D., Bloom J. W., Bailey T. J., Karp C. L., Halonen M., Martinez F. D., et al. A common single nucleotide polymorphism in the CD14 promoter decreases the affinity of Sp protein binding and enhances transcriptional activity. J Immunol 2001; 167: 5838–44
- Repo H., Anttonen K., Kilpinen S. K., Palotie A., Salven P., Orpana A., et al. CD14 and TNFa promoter polymorphisms in patients with acute arthritis. Special reference to development of chronic spondyloarthropathy. Scand J Rheumatol 2002; 31: 355–61
- Knijff‐Dutmer E., Drossaers‐Bakker W., Verhoeven A., van der Sluijs Veer G., Boers M., van der Linden S., et al. Rheumatoid factor measured by fluoroimmunoassay: a responsive measure of rheumatoid arthritis disease activity that is associated with joint damage. Ann Rheum Dis 2002; 61: 603–7