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Expert Opinion on Drug Metabolism & Toxicology Volume 15, 2019 - Issue 8
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Editorial

Is cytochrome oxidase inhibition the primary mechanism in aluminum phosphide poisoning?

Omid MehrpourRocky Mountain Poison and Drug Safety, Denver Health, Denver, CO, USACorrespondence[email protected]
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,
Natalie NeumannRocky Mountain Poison and Drug Safety, Denver Health, Denver, CO, USAView further author information
&
Patrick NgRocky Mountain Poison and Drug Safety, Denver Health, Denver, CO, USAView further author information
Pages 613-614 | Received 27 Mar 2019, Accepted 16 Jul 2019, Published online: 24 Jul 2019

References

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  • Dua R, Gill KD. Effect of aluminium phosphide exposure on kinetic properties of cytochrome oxidase and mitochondrial energy metabolism in rat brain. Biochim Biophys Acta. 2004 Sep 6;1674(1):4–11.
  • Cooper CE, Brown GC. The inhibition of mitochondrial cytochrome oxidase by the gases carbon monoxide, nitric oxide, hydrogen cyanide and hydrogen sulfide: chemical mechanism and physiological significance. J Bioenerg Biomembr. 2008 Oct;40(5):533–539.
  • Baud FJ, Haidar MK, Jouffroy R, et al. Determinants of lactic acidosis in acute cyanide poisonings. Crit Care Med. 2018 Jun;46(6):e523–e529.
  • Erfantalab P, Soltaninejad K, Shadnia S, et al. Trend of blood lactate level in acute aluminum phosphide poisoning. World J Emerg Med. 2017;8(2):116–120.
  • Mehrpour O, Jafarzadeh M, Abdollahi M. A systematic review of aluminium phosphide poisoning. Arh Hig Rada Toksikol. 2012 Mar;63(1):61–73.

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