REFERENCES
- Bulsara KR, Iskandar BJ, Villavicencio AT, Skene JH. A new millenium for spinal cord regeneration: growth-associated genes. Spine (Phila Pa 1976). 2002 Sep 1;27(17):1946–9.
- Szpara ML, Vranizan K, Tai YC, Goodman CS, Speed TP, Ngai J. Analysis of gene expression during neurite outgrowth and regeneration. BMC Neurosci. 2007;8:100.
- Busch SA, Silver J. The role of extracellular matrix in CNS regeneration. Curr Opin Neurobiol. 2007 Feb;17(1):120–7.
- Yiu G, He Z. Glial inhibition of CNS axon regeneration. Nat Rev Neurosci. 2006 Aug;7(8):617–27.
- Cafferty WB, Duffy P, Huebner E, Strittmatter SM. MAG and OMgp synergize with Nogo-A to restrict axonal growth and neurological recovery after spinal cord trauma. J Neurosci. 2010 May 19;30(20):6825–37.
- Giger RJ, Venkatesh K, Chivatakarn O, Raiker SJ, Robak L, Hofer T, Lee H, Rader C. Mechanisms of CNS myelin inhibition: evidence for distinct and neuronal cell type specific receptor systems. Restor Neurol Neurosci. 2008;26(2–3):97–115.
- Xie F, Zheng B. White matter inhibitors in CNS axon regeneration failure. Exp Neurol. 2008 Feb;209(2):302–12.
- Yamashita T, Fujitani M, Yamagishi S, Hata K, Mimura F. Multiple signals regulate axon regeneration through the Nogo receptor complex. Mol Neurobiol. 2005 Oct;32(2):105–11.
- Robak LA, Venkatesh K, Lee H, Raiker SJ, Duan Y, Lee-Osbourne J, Hofer T, Mage RG, Rader C, Giger RJ. Molecular basis of the interactions of the Nogo-66 receptor and its homolog NgR2 with myelin-associated glycoprotein: development of NgROMNI-Fc, a novel antagonist of CNS myelin inhibition. J Neurosci. 2009 May 6;29(18):5768–83.
- Lauren J, Hu F, Chin J, Liao J, Airaksinen MS, Strittmatter SM. Characterization of myelin ligand complexes with neuronal Nogo-66 receptor family members. J Biol Chem. 2007 Feb 23;282(8):5715–25.
- Venkatesh K, Chivatakarn O, Lee H, Joshi PS, Kantor DB, Newman BA, Mage R, Rader C, Giger R. The Nogo-66 receptor homolog NgR2 is a sialic acid-dependent receptor selective for myelin-associated glycoprotein. J Neurosci. 2005 Jan 26;25(4):808–22.
- Atwal JK, Pinkston-Gosse J, Syken J, Stawicki S, Wu Y, Shatz C, Tessier-Lavigne M. PirB is a functional receptor for myelin inhibitors of axonal regeneration. Science. 2008 Nov 7;322(5903):967–70.
- Fournier AE, GrandPre T, Strittmatter SM. Identification of a receptor mediating Nogo-66 inhibition of axonal regeneration. Nature. 2001 Jan 18;409(6818):341–6.
- Wang X, Chun SJ, Treloar H, Vartanian T, Greer CA, Strittmatter SM. Localization of Nogo-A and Nogo-66 receptor proteins at sites of axon-myelin and synaptic contact. J Neurosci. 2002 Jul 1;22(13):5505–15.
- Xu S, Liu M, Zhang T, Lv B, Liu B, Yuan W. Effect of lentiviral shRNA of Nogo receptor on rat cortex neuron axon outgrowth. Can J Neurol Sci. 2011 Jan;38(1):133–8.
- Woolf CJ, Bloechlinger S. Neuroscience. It takes more than two to Nogo. Science. 2002 Aug 16;297(5584):1132–4.
- Anson DS. The use of retroviral vectors for gene therapy—what are the risks? A review of retroviral pathogenesis and its relevance to retroviral vector-mediated gene delivery. Genet Vaccines Ther. 2004 Aug 13;2(1):9.
- Karolewski BA, Watson DJ, Parente MK, Wolfe JH. Comparison of transfection conditions for a lentivirus vector produced in large volumes. Hum Gene Ther. 2003 Sep 20;14(14):1287–96.
- Back SA, Luo NL, Borenstein NS, Levine JM, Volpe JJ, Kinney HC. Late oligodendrocyte progenitors coincide with the developmental window of vulnerability for human perinatal white matter injury. J Neurosci. 2001 Feb 15;21(4):1302–12.
- Davies JE, Tang X, Denning JW, Archibald SJ, Davies SJ. Decorin suppresses neurocan, brevican, phosphacan and NG2 expression and promotes axon growth across adult rat spinal cord injuries. Eur J Neurosci. 2004 Mar;19(5):1226–42.
- Polito A, Reynolds R. NG2-expressing cells as oligodendrocyte progenitors in the normal and demyelinated adult central nervous system. J Anat. 2005 Dec;207(6):707–16.
- Tan AM, Colletti M, Rorai AT, Skene JH, Levine JM. Antibodies against the NG2 proteoglycan promote the regeneration of sensory axons within the dorsal columns of the spinal cord. J Neurosci. 2006 May 3;26(18):4729–39.
- Tan AM, Petruska JC, Mendell LM, Levine JM. Sensory afferents regenerated into dorsal columns after spinal cord injury remain in a chronic pathophysiological state. Exp Neurol. 2007 Aug;206(2):257–68.