Correction

This article refers to:

MIR517C inhibits autophagy and the epithelial-to-mesenchymal (-like) transition phenotype in human glioblastoma through KPNA2-dependent disruption of TP53 nuclear translocation

Article title: MIR517C inhibits autophagy and the epithelial to mesenchymal(-like) transition phenotype in human glioblastoma through KPNA2-dependent disruption of TP53 nuclear translocation.

Authors: Yuntao Lu, Limin Xiao, Yawei Liu, Hai Wang, Hong Li, Qiang Zhou, Jun Pan, Bingxi Lei, Annie Huang, Songtao Qi

Journal: Autophagy

Bibliometrics: Volume 11, Issue 12, pages 2213-2232

DOI: https://doi.org/10.1080/15548627.2015.1108507.

In Figure 4D, the CDH13 band of western blot was mistakenly overlapped by the duplicate layer of the CLDN1 band when figure organization by using software, with resulting in the CDH13 band was not to be displayed. The authors are really sorry for the mistake. The corrected Fig. 4D is provided below:

Figure 4D: Western blotting was used to detect the expression of autophagy- and EMT-related proteins in U87 cells transiently transfected with ATG7 siRNA in normal medium or in the presence of TMZ.

In order to further confirm the result, the authors repeat the Western blot related to the Figure 4D. The result is provided as the Fig. 4D-R. And the result confirmed the EMT induced by TMZ was inhibited by siRNA of ATG7. Notably, some antibodies were different from those of Fig. 4D, so we also list the antibody information in the Tab. 1.

Figure 4D-R: The repeated Western blotting of the related proteins in Figure 4D.

Table 1. The antibody information and dilution for Western blot of Figure 4D-R.

Antibodies	SOURCE	CATALOG NO:	DILUTION
ATG7	Abcam	ab52472	WB 1:1000
CDH13	Santa Cruz Biotechnology	sc-166875	WB 1:1000
CDH2	Cell Signaling Technology	13116s	WB 1:1000
CTNNB1	Abcam	ab6302	WB 1:1000
CLDN1	Abcam	ab15098	WB 1:1000
SNAI1	Cell Signaling Technology	3879s	WB 1:1000
ZEB1	Cell Signaling Technology	3396s	WB 1:1000
LC3A/B	Cell Signaling Technology	12741s	WB 1:1000
ACTB	Cell Signaling Technology	4967s	WB 1:1000

The authors apologize for any inconvenience caused.