Host derived biomarkers of inflammation, apoptosis, and endothelial activation are associated with clinical outcomes in patients with bacteremia and sepsis regardless of microbial etiology

William O. HahnDivision of Allergy and Infectious Diseases, Department of Medicine, University of Washington, Seattle, WA, USACorrespondence[email protected]

Carmen MikacenicDivision of Pulmonary and Critical Care Medicine, Department of Medicine, University of Washington, Seattle, WA, USA

Brenda L. PriceDepartment of Biostatistics, University of Washington, Seattle, WA, USA

Susanna Harju-BakerDivision of Pulmonary and Critical Care Medicine, Department of Medicine, University of Washington, Seattle, WA, USA

Ronit KatzKidney Research Institute, University of Washington, Seattle, WA, USA

Jonathan HimmelfarbKidney Research Institute, University of Washington, Seattle, WA, USA

Mark M. WurfelDivision of Pulmonary and Critical Care Medicine, Department of Medicine, University of Washington, Seattle, WA, USA

W. Conrad LilesDivision of Allergy and Infectious Diseases, Department of Medicine, University of Washington, Seattle, WA, USA

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Disclosure of potential conflicts of interest

No potential conflicts of interest were disclosed.

Author contributions

WOH, CM, JH, MMW, and WCL designed the study. CM and SHB performed and analyzed the experiments. BLP and RK performed statistical analysis. WOH, MMH and WCL interpreted the results and drafted the manuscript. All authors reviewed, revised and approved the manuscript for submission.

Funding

Funding was provided by NIH T32AI007044-39 (WOH), NIH K23HL120896, and Parker B Francis Fellowship (CM), and Department of Medicine Research Fund (WCL).

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Host derived biomarkers of inflammation, apoptosis, and endothelial activation are associated with clinical outcomes in patients with bacteremia and sepsis regardless of microbial etiology

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Host derived biomarkers of inflammation, apoptosis, and endothelial activation are associated with clinical outcomes in patients with bacteremia and sepsis regardless of microbial etiology

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