MicroRNA-92a-3p-mediated inhibition of BCL11A upregulates γ-globin expression and inhibits oxidative stress and apoptosis in erythroid precursor cells

Huili Lia Department of Pediatrics, The First School of Clinical Medicine, Southern Medical University, Guangzhou, People’s Republic of China

Ruoping Linb Department of Pediatrics, Huizhou First Maternal and Child Health Care Hospital, Huizhou, People’s Republic of China

Huan Lic Department of Laboratory, Nanfang-Chunfu Children’s Institute of Hematology & Oncology, Dongguan, People’s Republic of China

Rilan Ouc Department of Laboratory, Nanfang-Chunfu Children’s Institute of Hematology & Oncology, Dongguan, People’s Republic of China

Kaiping Wangb Department of Pediatrics, Huizhou First Maternal and Child Health Care Hospital, Huizhou, People’s Republic of China

Junrong Linb Department of Pediatrics, Huizhou First Maternal and Child Health Care Hospital, Huizhou, People’s Republic of China

Chunfu Lia Department of Pediatrics, The First School of Clinical Medicine, Southern Medical University, Guangzhou, People’s Republic of ChinaCorrespondence[email protected]

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MicroRNA-92a-3p-mediated inhibition of BCL11A upregulates γ-globin expression and inhibits oxidative stress and apoptosis in erythroid precursor cells

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MicroRNA-92a-3p-mediated inhibition of BCL11A upregulates γ-globin expression and inhibits oxidative stress and apoptosis in erythroid precursor cells

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