Restricted intake of sulfur-containing amino acids reversed the hepatic injury induced by excess Desulfovibrio through gut–liver axis

Lingxi Zhoua The Key Laboratory of Industrial Biotechnology, Ministry of Education, School of Biotechnology, Jiangnan University, Wuxi, Jiangsu, ChinaView further author information

Gexue Lub Key Laboratory of Carbohydrate Chemistry and Biotechnology, Ministry of Education, School of Life Sciences and Health Engineering, Jiangnan University, Wuxi, ChinaView further author information

Yawen Nieb Key Laboratory of Carbohydrate Chemistry and Biotechnology, Ministry of Education, School of Life Sciences and Health Engineering, Jiangnan University, Wuxi, ChinaView further author information

Yilin Renc Department of Gastroenterology, Affiliated Hospital of Jiangnan University, Wuxi, ChinaView further author information

Jin-Song Shib Key Laboratory of Carbohydrate Chemistry and Biotechnology, Ministry of Education, School of Life Sciences and Health Engineering, Jiangnan University, Wuxi, ChinaView further author information

Yuzheng Xuec Department of Gastroenterology, Affiliated Hospital of Jiangnan University, Wuxi, ChinaView further author information

Zheng-Hong Xua The Key Laboratory of Industrial Biotechnology, Ministry of Education, School of Biotechnology, Jiangnan University, Wuxi, Jiangsu, China;d College of Biomass Science and Engineering, Sichuan University, Chengdu, ChinaCorrespondence[email protected]
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Yan Gengb Key Laboratory of Carbohydrate Chemistry and Biotechnology, Ministry of Education, School of Life Sciences and Health Engineering, Jiangnan University, Wuxi, China;c Department of Gastroenterology, Affiliated Hospital of Jiangnan University, Wuxi, ChinaCorrespondence[email protected]

https://orcid.org/0000-0001-8861-7686 View further author information

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Restricted intake of sulfur-containing amino acids reversed the hepatic injury induced by excess Desulfovibrio through gut–liver axis

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Restricted intake of sulfur-containing amino acids reversed the hepatic injury induced by excess Desulfovibrio through gut–liver axis

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