Cerebrospinal fluid viral load is related to cortical atrophy and not to intracerebral calcifications in children with symptomatic HIV disease

Pirn Brouwers HIV & AIDS Malignancy Branch, National Cancer Institute, Bethesda, MD, 20892, USA

Lucy Civitello HIV & AIDS Malignancy Branch, National Cancer Institute, Bethesda, MD, 20892, USA; Children's Hospital Medical Cente, Washington, DC, 20010, USA

Charles Decarli Epilepsy Research Branch, National Institute of Neurological Disorders & Stroke, Bethesda, MD, 20892, USA

Pamela Wolters HIV & AIDS Malignancy Branch, National Cancer Institute, Bethesda, MD, 20892, USA; Medical Illness Counseling Center, Chevy Chase, MD, 20814, USA

Shizuko Sei HIV & AIDS Malignancy Branch, National Cancer Institute, Bethesda, MD, 20892, USA

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Renee Smith & Megan Wilkins. (2015) Perinatally acquired HIV infection: Long-term neuropsychological consequences and challenges ahead. Child Neuropsychology 21:2, pages 234-268.
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L. Calza, R. Manfredi, E. Freo, B. Farneti, L. Tampellini, G. d'orsi & F. Chiodo. (2004) Transient Reversal of HIV-Associated Motor Neuron Disease Following the Introduction of Highly Active Antiretroviral Therapy. Journal of Chemotherapy 16:1, pages 98-101.
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EMMANUEL G. RÉGULIER, KRYZSZTOF REISS, KAMEL KHALILI, SHOHREH AMINI, JAY RAPPAPORT, JEAN-FRANÇOIS ZAGURY & PETER D. KATSIKIS. (2004) T-CELL AND NEURONAL APOPTOSIS IN HIV INFECTION: IMPLICATIONS FOR THERAPEUTIC INTERVENTION. International Reviews of Immunology 23:1-2, pages 25-59.
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Cerebrospinal fluid viral load is related to cortical atrophy and not to intracerebral calcifications in children with symptomatic HIV disease

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Cerebrospinal fluid viral load is related to cortical atrophy and not to intracerebral calcifications in children with symptomatic HIV disease

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