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Cancer Biology & Therapy Volume 15, 2014 - Issue 6
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Research Paper

MALAT1 promotes the proliferation and metastasis of gallbladder cancer cells by activating the ERK/MAPK pathway

Xiang-Song Wu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Xu-An Wang Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Wen-Guang Wu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Yun-Ping Hu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Mao-Lan Li Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Qian Ding Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Hao Weng Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
,
Yi-Jun Shu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Tian-Yu Liu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Lin Jiang Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Yang Cao Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
,
Run-Fa Bao Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Jia-Sheng Mu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
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Zhu-Jun Tan Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China
,
Feng Tao Department of Gastrointestinal Surgery; Shaoxing People’s Hospital; Shaoxing, PR ChinaCorrespondence[email protected] [email protected]
&
Ying-Bin Liu Department of General Surgery; Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Laboratory of General Surgery, Xinhua Hospital; Shanghai Jiao Tong University School of Medicine; Shanghai, PR China; Institute of Biliary Tract Disease; Shanghai Jiao Tong University School of Medicine; Shanghai, PR ChinaCorrespondence[email protected] [email protected]
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Pages 806-814 | Received 23 Dec 2013, Accepted 18 Mar 2014, Published online: 21 Mar 2014

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Cheng Peng, Yuchi Wang, Liyang Ji, Liangju Kuang, Ziyan Yu, Hanrong Li, Jinsong Zhang & Jiangyue Zhao. (2021) LncRNA-MALAT1/miRNA-204-5p/Smad4 Axis Regulates Epithelial–Mesenchymal Transition, Proliferation and Migration of Lens Epithelial Cells. Current Eye Research 46:8, pages 1137-1147.
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Fujun Yu, Zhongqiu Lu, Jing Cai, Kate Huang, Bicheng Chen, Guojun Li, Peihong Dong & Jianjian Zheng. (2015) MALAT1 functions as a competing endogenous RNA to mediate Rac1 expression by sequestering miR-101b in liver fibrosis. Cell Cycle 14:24, pages 3885-3896.
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