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DNA damage causes TP53-dependent coupling of self-renewal and senescence pathways in embryonal carcinoma cells

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Pages 430-441 | Published online: 01 Feb 2012

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Mikhail Baryshev, Inna Inashkina, Kristine Salmina, Anda Huna, Thomas R Jackson & Jekaterina Erenpreisa. (2018) DNA methylation of the Oct4A enhancers in embryonal carcinoma cells after etoposide treatment is associated with alternative splicing and altered pluripotency in reversibly senescent cells. Cell Cycle 17:3, pages 362-366.
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Kristine Salmina, Anda Huna, Inna Inashkina, Alexander Belyayev, Jekabs Krigerts, Ladislava Pastova, Alejandro Vazquez-Martin & Jekaterina Erenpreisa. (2017) Nucleolar aggresomes mediate release of pericentric heterochromatin and nuclear destruction of genotoxically treated cancer cells. Nucleus 8:2, pages 205-221.
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Anda Huna, Kristine Salmina, Jekaterina Erenpreisa, Alejandro Vazquez-Martin, Jekabs Krigerts, Inna Inashkina, Bogdan I Gerashchenko, Paul A Townsend, Mark S Cragg & Thomas R Jackson. (2015) Role of stress-activated OCT4A in the cell fate decisions of embryonal carcinoma cells treated with etoposide . Cell Cycle 14:18, pages 2969-2984.
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Tatyana V. Pospelova, Tatiana V Bykova, Svetlana G Zubova, Natalia V Katolikova, Natalia M Yartzeva & Valery A Pospelov. (2013) Rapamycin induces pluripotent genes associated with avoidance of replicative senescence. Cell Cycle 12:24, pages 3841-3851.
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A O Pisco & S Huang. (2015) Non-genetic cancer cell plasticity and therapy-induced stemness in tumour relapse: ‘What does not kill me strengthens me’. British Journal of Cancer 112:11, pages 1725-1732.
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Sui Huang. (2014) The War on Cancer: Lessons from the War on Terror. Frontiers in Oncology 4.
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