B cell intrinsic expression of IFNλ receptor suppresses the acute humoral immune response to experimental blood-stage malaria

William O. Hahna Division of Allergy and Infectious Diseases, Department of Medicine, University of Washington, Seattle, USA

Marion Pepperb Department of Immunology, University of Washington, Seattle, USA

W. Conrad Lilesa Division of Allergy and Infectious Diseases, Department of Medicine, University of Washington, Seattle, USACorrespondence[email protected]
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Figures & data

Figure 1. Absence of interferon lambda leads to improved parasite control during blood-stage malaria infection.

Figure 2. Absence of interferon lambda leads to increased antibody titers and increased plasmablast numbers.

Figure 3. Absence of interferon lambda leads to increased CD4+ T effector cells.

Figure 4. Interferon lambda signals suppress plasmablast formation in a B cell-intrinsic fashion.

Figure 5. Improved control of blood-stage infection with absence of Interferon lambda-specific B cells.

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