Diabetes and the heart: could the diabetic myocardium be protected by preconditioning?

Vladimir VinokurDepartment of Cellular Biochemistry and Human GeneticsThe Hebrew University of Jerusalem, Jerusalem, Israel; Department of Otolaryngology, The Hadassah-Hebrew University Hospital, Jerusalem, Israel

Gil LeibowitzDepartment of EndocrinologyThe Hadassah-Hebrew University Hospital, Jerusalem, Israel

Leonid GrinbergDepartment of Cellular Biochemistry and Human GeneticsThe Hebrew University of Jerusalem, Jerusalem, Israel

Ron EliasharDepartment of OtolaryngologyThe Hadassah-Hebrew University Hospital, Jerusalem, Israel

Eduard BerenshteinDepartment of Cellular Biochemistry and Human GeneticsThe Hebrew University of Jerusalem, Jerusalem, Israel

Mordechai ChevionDepartment of Cellular Biochemistry and Human GeneticsThe Hebrew University of Jerusalem, Jerusalem, Israel

Abstract

Both type 1 and type 2 diabetes (insulin-dependent and non-insulin dependent diabetes, respectively) are associated with increased risk for microvascular and macrovascular complications including retinopathy, neuropathy, nephropathy and atherosclerosis. Type 2 diabetes markedly increases the risk for cardiovascular morbidity and mortality, which has major public health implications. In this review, molecular mechanisms pertaining to diabetes-induced heart pathology are addressed.

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Diabetes and the heart: could the diabetic myocardium be protected by preconditioning?

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Diabetes and the heart: could the diabetic myocardium be protected by preconditioning?

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