T-cell-dependent mechanisms promote Ebola VLP-induced antibody responses, but are dispensable for vaccine-mediated protection

Protection from EBOV in the absence of antibodies

Christopher L CooperMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USACorrespondence[email protected]

Karen A MartinsMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USA

Sabrina M StronskyMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USA

David P LanganMolecular Microbiology and ImmunologyUniversity of Maryland School of Medicine, Baltimore, MD21201, USA

Jesse SteffensMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USA

Sean Van TongerenMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USA

Sina BavariMolecular and Translational Sciences, United States Army Medical Research Institute of Infectious Diseases, Frederick, MD21702, USA

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T-cell-dependent mechanisms promote Ebola VLP-induced antibody responses, but are dispensable for vaccine-mediated protection

Protection from EBOV in the absence of antibodies

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T-cell-dependent mechanisms promote Ebola VLP-induced antibody responses, but are dispensable for vaccine-mediated protection

Protection from EBOV in the absence of antibodies

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