New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: Inclusions containing SOD1 in neurons and astrocytes

Shinsuke Kato Division of Neuropathology, Faculty of Medicine, Tottori University, Yonago, Japan

Miki Takikawa Division of Neuropathology, Faculty of Medicine, Tottori University, Yonago, Japan

Kenji Nakashima Division of Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, Yonago, Japan

Asao Hirano Division of Neuropathology, Montefiore Medical Center, New York, USA

Don W Cleveland Ludwig Institute for Cancer Research and Departments of Medicine and Neurosciences, University of California at San Diego, San Diego, USA

Hirofumi Kusaka Department of Neurology, Kansai Medical University, Moriguchi, Japan

Noriyuki Shibata Department of Pathology, Tokyo Women's Medical College, Tokyo, Japan

Masako Kato Secon Department of Pathology, Faculty of Medicine, Tottori University, Yonago, Japan

Imaharu Nakano Department of Neurology, Jichi Medical College, Tochigi, Japan

Eisaku Ohama Division of Neuropathology, Faculty of Medicine, Tottori University, Yonago, Japan

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New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: Inclusions containing SOD1 in neurons and astrocytes

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New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: Inclusions containing SOD1 in neurons and astrocytes

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