Sustained protein synthesis and reduced eEF2K levels in TAp73^-\- mice brain: a possible compensatory mechanism

Barak RotblatMRC Toxicology Unit, University of Cambridge, Rome, UK;Department of Life Sciences, Ben Gurion University in the Negev, Beer Sheva, IsraelCorrespondence[email protected] [email protected] [email protected]

http://orcid.org/0000-0003-2985-7115

Massimiliano AgostiniMRC Toxicology Unit, University of Cambridge, Rome, UK;Department of Experimental Medicine and Surgery, IDI-IRCCS, University of Rome Tor Vergata, Rome, Italy

http://orcid.org/0000-0003-3124-2072

Maria Victoria Niklison-ChirouMRC Toxicology Unit, University of Cambridge, Rome, UK;Blizard Institute of Cell and Molecular Science, Barts and the London School of Medicine and Dentistry, Queen Mary University of London, London, UK

Ivano AmelioMRC Toxicology Unit, University of Cambridge, Rome, UK

Anne E. WillisMRC Toxicology Unit, University of Cambridge, Rome, UKCorrespondence[email protected] [email protected] [email protected]

Gerry MelinoMRC Toxicology Unit, University of Cambridge, Rome, UK;Department of Experimental Medicine and Surgery, IDI-IRCCS, University of Rome Tor Vergata, Rome, ItalyCorrespondence[email protected] [email protected] [email protected]

Sustained protein synthesis and reduced eEF2K levels in TAp73^-\- mice brain: a possible compensatory mechanism

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Sustained protein synthesis and reduced eEF2K levels in TAp73-\- mice brain: a possible compensatory mechanism

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Sustained protein synthesis and reduced eEF2K levels in TAp73^-\- mice brain: a possible compensatory mechanism