Methyl-β-cyclodextrin restores impaired autophagy flux in Niemann-Pick C1-deficient cells through activation of AMPK

Sheng Daia National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USA;b Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, Hangzhou, China

Andrés E. Dulceya National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USA

Xin Hua National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USA

Christopher A. Wassifc National Institute of Child Health and Human Development, NIH, Bethesda, MD, USA

Forbes D. Porterc National Institute of Child Health and Human Development, NIH, Bethesda, MD, USA

Christopher P. Austina National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USA

Daniel S. Oryd Diabetic Cardiovascular Disease Center, Washington University School of Medicine, St. Louis, MOUSACorrespondence[email protected] [email protected] [email protected]

Juan Marugana National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USACorrespondence[email protected] [email protected] [email protected]

Wei Zhenga National Center for Advancing Translational Sciences (NCATS), NIH, Bethesda, MD, USACorrespondence[email protected] [email protected] [email protected]

https://orcid.org/0000-0003-1034-0757

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Methyl-β-cyclodextrin restores impaired autophagy flux in Niemann-Pick C1-deficient cells through activation of AMPK

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Methyl-β-cyclodextrin restores impaired autophagy flux in Niemann-Pick C1-deficient cells through activation of AMPK

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